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Registros recuperados : 5 | |
1. |  | GUIMARÃES, L. M. da S.; TITON, M.; LAU, D.; ROSSE, L. N.; OLIVEIRA, L. S. S.; ROSADO, C. C. G.; CHRISTO, G. G. O.; ALFENAS, A. C. Eucalyptus pellita as a source of resistance to rust, ceratocystis wilt and leaf blight. Crop Breeding and Applied Biotechnology, Londrina, v. 10, n. 2, p. 124-131, June 2010. Biblioteca(s): Embrapa Agricultura Digital. |
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2. |  | ROSADO, C. C. G.; GUIMARÃES, L. M. da S.; FARIA, D. A.; RESENDE, M. D. V. de; CRUZ, C. D.; GRATTAPAGLIA, D.; ALFENAS, A. C. QTL mapping for resistance to Ceratocystis wilt in Eucalyptus. Tree Genetics & Genomes, v. 12, n. 4, 10 p., 2016. Biblioteca(s): Embrapa Florestas; Embrapa Recursos Genéticos e Biotecnologia. |
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3. |  | ROSADO, C. C. G.; GUIMARÃES, L. M. da S.; TITON, M.; LAU, D.; ROSSE, L.; RESENDE, M. D. V. de; ALFENAS, A. C. Resistance to ceratocystis wilt (Ceratocystis fimbriata) in parents and progenies of Eucalyptus grandis x E. urophylla. Silvae Genetica, v. 59, n. 2/3, p. 99-106, 2010. Biblioteca(s): Embrapa Florestas. |
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5. |  | ALVES, A. A.; ROSADO, C. C. G.; FARIA, D. A.; GUIMARÃES, L. M. da S.; LAU, D.; BROMMONSCHENKEL, S. H.; GRATTAPAGLIA, D.; ALFENAS, A. C. Genetic mapping provides evidence for the role of additive and non-additive QTLs in the response of inter-specific hybrids of Eucalyptus to Puccinia psidii rust infection. Euphytica, Wageningen, v. 183, n. 1, p. 27-38, Jan. 2012. Biblioteca(s): Embrapa Agroenergia; Embrapa Recursos Genéticos e Biotecnologia; Embrapa Trigo. |
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Registros recuperados : 5 | |
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Registro Completo
Biblioteca(s): |
Embrapa Florestas; Embrapa Recursos Genéticos e Biotecnologia. |
Data corrente: |
30/09/2016 |
Data da última atualização: |
25/04/2024 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
A - 2 |
Autoria: |
ROSADO, C. C. G.; GUIMARÃES, L. M. da S.; FARIA, D. A.; RESENDE, M. D. V. de; CRUZ, C. D.; GRATTAPAGLIA, D.; ALFENAS, A. C. |
Afiliação: |
CARLA CRISTINA GONÇALVES ROSADO, UFV; LÚCIO MAURO DA SILVA GUIMARÃES, UFV; DANIELLE ASSIS FARIA; MARCOS DEON VILELA DE RESENDE, CNPF; COSME DAMIÃO CRUZ, UFV; DARIO GRATTAPAGLIA, CENARGEN; ACELINO COUTO ALFENAS, UFV. |
Título: |
QTL mapping for resistance to Ceratocystis wilt in Eucalyptus. |
Ano de publicação: |
2016 |
Fonte/Imprenta: |
Tree Genetics & Genomes, v. 12, n. 4, 10 p., 2016. |
DOI: |
10.1007/s11295-016-1029-4 |
Idioma: |
Inglês |
Conteúdo: |
Ceratocystis wilt caused by the fungus Ceratocystis fimbriata, is currently one of the major diseases in commercial plantations of Eucalyptus trees in Brazil. Deployment of resistant genotypes has been the main strategy for effective disease management. The present study aimed at identifying genomic regions underlying the genetic control of resistance to Ceratocystis wilt in Eucalyptus by quantitative trait loci (QTL) mapping in an outbred hybrid progeny derived from a cross between (Eucalyptus dunnii × Eucalyptus grandis) × (Eucalyptus urophylla × Eucalyptus globulus). A segregating population of 127 individuals was phenotyped for resistance to Ceratocystis wilt using controlled inoculation under a completely randomized design with five clonal replicates per individual plant. The phenotypic resistance response followed a continuous variation, enabling us to analyze the trait in a quantitative manner. The population was genotyped with 114 microsatellite markers and 110 were mapped with an average interval of 12.3 cM. Using a sib-pair interval-mapping approach five QTLs were identified for disease resistance, located on linkage groups 1, 3, 5, 8, and 10, and their estimated individual heritability ranged from 0.096 to 0.342. The QTL on linkage group 3 overlaps with other fungal disease-resistance QTLs mapped earlier and is consistent with the annotation of several disease-resistance genes on this chromosome in the E. grandis genome. This is the first study to identify and attempt to quantify the effects of QTLs associated with resistance to Ceratocystis wilt in Eucalyptus. MenosCeratocystis wilt caused by the fungus Ceratocystis fimbriata, is currently one of the major diseases in commercial plantations of Eucalyptus trees in Brazil. Deployment of resistant genotypes has been the main strategy for effective disease management. The present study aimed at identifying genomic regions underlying the genetic control of resistance to Ceratocystis wilt in Eucalyptus by quantitative trait loci (QTL) mapping in an outbred hybrid progeny derived from a cross between (Eucalyptus dunnii × Eucalyptus grandis) × (Eucalyptus urophylla × Eucalyptus globulus). A segregating population of 127 individuals was phenotyped for resistance to Ceratocystis wilt using controlled inoculation under a completely randomized design with five clonal replicates per individual plant. The phenotypic resistance response followed a continuous variation, enabling us to analyze the trait in a quantitative manner. The population was genotyped with 114 microsatellite markers and 110 were mapped with an average interval of 12.3 cM. Using a sib-pair interval-mapping approach five QTLs were identified for disease resistance, located on linkage groups 1, 3, 5, 8, and 10, and their estimated individual heritability ranged from 0.096 to 0.342. The QTL on linkage group 3 overlaps with other fungal disease-resistance QTLs mapped earlier and is consistent with the annotation of several disease-resistance genes on this chromosome in the E. grandis genome. This is the first study to identify and att... Mostrar Tudo |
Palavras-Chave: |
Genetic mapping; Sib-pair interval-mapping. |
Thesagro: |
Ceratocystis Fimbriata; Doença de planta; Fungo. |
Thesaurus NAL: |
Ceratocystis; Eucalyptus; Plant diseases and disorders; quantitative trait loci. |
Categoria do assunto: |
-- G Melhoramento Genético |
URL: |
https://www.alice.cnptia.embrapa.br/alice/bitstream/doc/1053783/1/Rosado2016ArticleQTLMappingForResistanceToCerat.pdf
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Marc: |
LEADER 02518naa a2200313 a 4500 001 2053783 005 2024-04-25 008 2016 bl uuuu u00u1 u #d 024 7 $a10.1007/s11295-016-1029-4$2DOI 100 1 $aROSADO, C. C. G. 245 $aQTL mapping for resistance to Ceratocystis wilt in Eucalyptus.$h[electronic resource] 260 $c2016 520 $aCeratocystis wilt caused by the fungus Ceratocystis fimbriata, is currently one of the major diseases in commercial plantations of Eucalyptus trees in Brazil. Deployment of resistant genotypes has been the main strategy for effective disease management. The present study aimed at identifying genomic regions underlying the genetic control of resistance to Ceratocystis wilt in Eucalyptus by quantitative trait loci (QTL) mapping in an outbred hybrid progeny derived from a cross between (Eucalyptus dunnii × Eucalyptus grandis) × (Eucalyptus urophylla × Eucalyptus globulus). A segregating population of 127 individuals was phenotyped for resistance to Ceratocystis wilt using controlled inoculation under a completely randomized design with five clonal replicates per individual plant. The phenotypic resistance response followed a continuous variation, enabling us to analyze the trait in a quantitative manner. The population was genotyped with 114 microsatellite markers and 110 were mapped with an average interval of 12.3 cM. Using a sib-pair interval-mapping approach five QTLs were identified for disease resistance, located on linkage groups 1, 3, 5, 8, and 10, and their estimated individual heritability ranged from 0.096 to 0.342. The QTL on linkage group 3 overlaps with other fungal disease-resistance QTLs mapped earlier and is consistent with the annotation of several disease-resistance genes on this chromosome in the E. grandis genome. This is the first study to identify and attempt to quantify the effects of QTLs associated with resistance to Ceratocystis wilt in Eucalyptus. 650 $aCeratocystis 650 $aEucalyptus 650 $aPlant diseases and disorders 650 $aquantitative trait loci 650 $aCeratocystis Fimbriata 650 $aDoença de planta 650 $aFungo 653 $aGenetic mapping 653 $aSib-pair interval-mapping 700 1 $aGUIMARÃES, L. M. da S. 700 1 $aFARIA, D. A. 700 1 $aRESENDE, M. D. V. de 700 1 $aCRUZ, C. D. 700 1 $aGRATTAPAGLIA, D. 700 1 $aALFENAS, A. C. 773 $tTree Genetics & Genomes$gv. 12, n. 4, 10 p., 2016.
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Embrapa Recursos Genéticos e Biotecnologia (CENARGEN) |
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