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Registro Completo |
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Biblioteca(s): |
Embrapa Unidades Centrais. |
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Data corrente: |
05/02/2018 |
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Data da última atualização: |
05/02/2018 |
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Autoria: |
ARAUJO, J. L. de; CRISTO, T. G. de; MORAIS, R. M. de; COSTA, L. S. da; BIEZUS, G.; MULLER, T. R.; RECH, R. R.; CASAGRANDE, R. A. |
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Afiliação: |
Jeann Leal de Araujo, Department of Veterinary Pathobiology/Texas A&M University; Thierry Grima de Cristo, LPA/CCA/UDESC; Raissa Moreira de Morais, LPA/CCA/UDESC; Leonardo Silva da Costa, LPA/CCA/UDESC; Giovana Biezus; Thiago Rinaldi Müller, Hospital de Clínicas Veterinárias/Centro de Ciências Agroveterinárias/UDESC; Raquel Rubia Rech, Department of Veterinary Pathobiology/Texas A&M University; Renata Assis Casagrande, LPA/CCA/UDESC. |
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Título: |
Proventricular dilatation disease (PDD) outbreak in blue-and-gold macaws (Ara ararauna) in the State of Santa Catarina, southern Brazil. |
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Ano de publicação: |
2017 |
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Fonte/Imprenta: |
Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 37, n. 11, p. 1331-1335, novembro. 2017. |
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Idioma: |
Inglês |
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Notas: |
Título em português: Surto de doença da dilatação proventricular em araras-canindé (Ara ararauna) no estado de Santa Catarina, sul do Brasil. |
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Conteúdo: |
Proventricular dilatation disease (PDD) is a lethal and important disease of captive psittacine birds, and affects a wide range of species, including endangered ones, and lacks an effective treatment. This report describes PDD in three blue-and-gold macaws (Ara ararauna) in southern Brazil. All three macaws originated from the same aviary and presented similar clinical signs including anorexia, apathy, emaciation and prostration. At necropsy, one of the macaws presented an enlarged proventriculus. Histologically, ymphoplasmacytic infiltrates was observed in the ganglia and nerves of the esophagus, crop, proventriculus, ventriculus, heart, adrenal glands, and adrenal medulla of all three cases. Two macaws had meningoencephalomyelitis and one had myocarditis. Immunohistochemistry identified PaBV antigen in the brain, proventricular, ventricular ganglia, and epicardial ganglia, and cardiomyocytes of all three macaws. |
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Palavras-Chave: |
Arara-canindé; Blue-and-gold macaw; Doença da dilatação proventricular; Doença viral; Ganglioneurite; Ganglioneuritis; Meningoencefalomielite; PDD; Proventricular dilatation disease; Psitacídeos; Psittacine birds. |
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Thesaurus Nal: |
Ara ararauna; Meningoencephalitis; Viral diseases of animals and humans. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/172159/1/Proventricular-dilatation-disease-PDD.pdf
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Marc: |
LEADER 02290naa a2200385 a 4500
001 2087060
005 2018-02-05
008 2017 bl uuuu u00u1 u #d
100 1 $aARAUJO, J. L. de
245 $aProventricular dilatation disease (PDD) outbreak in blue-and-gold macaws (Ara ararauna) in the State of Santa Catarina, southern Brazil.$h[electronic resource]
260 $c2017
500 $aTítulo em português: Surto de doença da dilatação proventricular em araras-canindé (Ara ararauna) no estado de Santa Catarina, sul do Brasil.
520 $aProventricular dilatation disease (PDD) is a lethal and important disease of captive psittacine birds, and affects a wide range of species, including endangered ones, and lacks an effective treatment. This report describes PDD in three blue-and-gold macaws (Ara ararauna) in southern Brazil. All three macaws originated from the same aviary and presented similar clinical signs including anorexia, apathy, emaciation and prostration. At necropsy, one of the macaws presented an enlarged proventriculus. Histologically, ymphoplasmacytic infiltrates was observed in the ganglia and nerves of the esophagus, crop, proventriculus, ventriculus, heart, adrenal glands, and adrenal medulla of all three cases. Two macaws had meningoencephalomyelitis and one had myocarditis. Immunohistochemistry identified PaBV antigen in the brain, proventricular, ventricular ganglia, and epicardial ganglia, and cardiomyocytes of all three macaws.
650 $aAra ararauna
650 $aMeningoencephalitis
650 $aViral diseases of animals and humans
653 $aArara-canindé
653 $aBlue-and-gold macaw
653 $aDoença da dilatação proventricular
653 $aDoença viral
653 $aGanglioneurite
653 $aGanglioneuritis
653 $aMeningoencefalomielite
653 $aPDD
653 $aProventricular dilatation disease
653 $aPsitacídeos
653 $aPsittacine birds
700 1 $aCRISTO, T. G. de
700 1 $aMORAIS, R. M. de
700 1 $aCOSTA, L. S. da
700 1 $aBIEZUS, G.
700 1 $aMULLER, T. R.
700 1 $aRECH, R. R.
700 1 $aCASAGRANDE, R. A.
773 $tPesquisa Veterinária Brasileira, Rio de Janeiro$gv. 37, n. 11, p. 1331-1335, novembro. 2017.
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Registro original: |
Embrapa Unidades Centrais (AI-SEDE) |
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Registro Completo
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Biblioteca(s): |
Embrapa Agricultura Digital. |
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Data corrente: |
21/11/2012 |
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Data da última atualização: |
14/04/2020 |
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Tipo da produção científica: |
Artigo em Periódico Indexado |
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Circulação/Nível: |
A - 1 |
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Autoria: |
PROTASIO, A. V.; TSAI, I. J.; BABBAGE, A.; NICHOL, S.; HUNT, M.; ASLETT, M. A.; SILVA, N. de; VELARDE, G. S.; ANDERSON, T. J. C.; CLARK, R. C.; DAVIDSON, C.; DILLON, G. P.; HOLROYD, N. E.; LOVERDE, P. T.; LLOYD, C.; MCQUILLAN, J.; OLIVEIRA, G.; OTTO, T. D.; PARKER-MANUEL, S. J.; QUAIL, M. A.; WILSON, R. A.; ZERLOTINI, A.; DUNNE, D. W.; BERRIMAN, M. |
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Afiliação: |
ANNA V. PROTASIO, Wellcome Trust Sanger Institute; ISHENG J. TSAI, Wellcome Trust Sanger Institute; ANNE BABBAGE, Wellcome Trust Sanger Institute; SARAH NICHOL, Wellcome Trust Sanger Institute; MARTIN HUNT, Wellcome Trust Sanger Institute; MARTIN A. ASLETT, Wellcome Trust Sanger Institute; NISHADI DE SILVA, Wellcome Trust Sanger Institute; GILES S. VELARDE, Wellcome Trust Sanger Institute; TIM J. C. ANDERSON, Texas Biomedical Research Institute; RICHARD C. CLARK, Wellcome Trust Sanger Institute; CLAIRE DAVIDSON, Wellcome Trust Sanger Institute; GARY P. DILLON, Wellcome Trust Sanger Institute; NANCY E. HOLROYD, Wellcome Trust Sanger Institute; PHILIP T. LOVERDE, University of Texas Health Science Center; CHRISTINE LLOYD, Wellcome Trust Sanger Institute; JACQUELLINE MCQUILLAN, Wellcome Trust Sanger Institute; GUILHERME OLIVEIRA, Centro de Pesquisas René Rachou, Fundação Oswaldo Cruz; THOMAS D. OTTO, Wellcome Trust Sanger Institute; SOPHIA J. PARKER-MANUEL, University of York; MICHAEL A. QUAIL, Wellcome Trust Sanger Institute; R. ALAN WILSON, University of York; ADHEMAR ZERLOTINI NETO, CNPTIA; DAVID W. DUNNE, University of Cambridge; MATTHEW BERRIMAN, Wellcome Trust Sanger Institute. |
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Título: |
A systematically improved high quality genome and transcriptome of the human blood fluke Schistosoma mansoni. |
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Ano de publicação: |
2012 |
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Fonte/Imprenta: |
PLOS Neglected Tropical Diseases, v. 6, n. 1, p. 1-13, Jan. 2012. |
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DOI: |
10.1371/journal.pntd.0001455 |
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Idioma: |
Inglês |
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Conteúdo: |
Schistosomiasis is one of the most prevalent parasitic diseases, affecting millions of people in developing countries. Amongst the human-infective species, Schistosoma mansoni is also the most commonly used in the laboratory and here we present the systematic improvement of its draft genome. We used Sanger capillary and deep-coverage Illumina sequencing from clonal worms to upgrade the highly fragmented draft 380 Mb genome to one with only 885 scaffolds and more than 81% of the bases organised into chromosomes. We have also used transcriptome sequencing (RNA-seq) from four time points in the parasite?s life cycle to refine gene predictions and profile their expression. More than 45% of predicted genes have been extensively modified and the total number has been reduced from 11,807 to 10,852. Using the new version of the genome, we identified trans-splicing events occurring in at least 11% of genes and identified clear cases where it is used to resolve polycistronic transcripts. We have produced a high-resolution map of temporal changes in expression for 9,535 genes, covering an unprecedented dynamic range for this organism. All of these data have been consolidated into a searchable format within the GeneDB (www.genedb.org) and SchistoDB (www.schistodb.net) databases. With further transcriptional profiling and genome sequencing increasingly accessible, the upgraded genome will form a fundamental dataset to underpin further advances in schistosome research. |
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Thesaurus NAL: |
Schistosoma. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/70531/1/journal.pntd.0001455.pdf
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Marc: |
LEADER 02660naa a2200421 a 4500
001 1940202
005 2020-04-14
008 2012 bl uuuu u00u1 u #d
024 7 $a10.1371/journal.pntd.0001455$2DOI
100 1 $aPROTASIO, A. V.
245 $aA systematically improved high quality genome and transcriptome of the human blood fluke Schistosoma mansoni.$h[electronic resource]
260 $c2012
520 $aSchistosomiasis is one of the most prevalent parasitic diseases, affecting millions of people in developing countries. Amongst the human-infective species, Schistosoma mansoni is also the most commonly used in the laboratory and here we present the systematic improvement of its draft genome. We used Sanger capillary and deep-coverage Illumina sequencing from clonal worms to upgrade the highly fragmented draft 380 Mb genome to one with only 885 scaffolds and more than 81% of the bases organised into chromosomes. We have also used transcriptome sequencing (RNA-seq) from four time points in the parasite?s life cycle to refine gene predictions and profile their expression. More than 45% of predicted genes have been extensively modified and the total number has been reduced from 11,807 to 10,852. Using the new version of the genome, we identified trans-splicing events occurring in at least 11% of genes and identified clear cases where it is used to resolve polycistronic transcripts. We have produced a high-resolution map of temporal changes in expression for 9,535 genes, covering an unprecedented dynamic range for this organism. All of these data have been consolidated into a searchable format within the GeneDB (www.genedb.org) and SchistoDB (www.schistodb.net) databases. With further transcriptional profiling and genome sequencing increasingly accessible, the upgraded genome will form a fundamental dataset to underpin further advances in schistosome research.
650 $aSchistosoma
700 1 $aTSAI, I. J.
700 1 $aBABBAGE, A.
700 1 $aNICHOL, S.
700 1 $aHUNT, M.
700 1 $aASLETT, M. A.
700 1 $aSILVA, N. de
700 1 $aVELARDE, G. S.
700 1 $aANDERSON, T. J. C.
700 1 $aCLARK, R. C.
700 1 $aDAVIDSON, C.
700 1 $aDILLON, G. P.
700 1 $aHOLROYD, N. E.
700 1 $aLOVERDE, P. T.
700 1 $aLLOYD, C.
700 1 $aMCQUILLAN, J.
700 1 $aOLIVEIRA, G.
700 1 $aOTTO, T. D.
700 1 $aPARKER-MANUEL, S. J.
700 1 $aQUAIL, M. A.
700 1 $aWILSON, R. A.
700 1 $aZERLOTINI, A.
700 1 $aDUNNE, D. W.
700 1 $aBERRIMAN, M.
773 $tPLOS Neglected Tropical Diseases$gv. 6, n. 1, p. 1-13, Jan. 2012.
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Embrapa Agricultura Digital (CNPTIA) |
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