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Registro Completo |
Biblioteca(s): |
Embrapa Soja. |
Data corrente: |
05/10/2000 |
Data da última atualização: |
05/10/2000 |
Autoria: |
JUNG, R.; TOLEDO, J. F. F. de; ARIAS, C. A. A.; YORINORI, J. T. |
Título: |
Resistencia da cultivar FT-2 a mancha parda (Septoria glycines) avaliadas em geracoes F2 e F3 em soja. |
Ano de publicação: |
2000 |
Fonte/Imprenta: |
Genetics and Molecular Biology, Ribeirao Preto, v.23, n.3, p.547, Sept. 2000. Supplement. |
Idioma: |
Português |
Notas: |
Trabalho apresentado no 46, Congresso Nacional de Genetica, 19 a 23 de setembro de 2000, Aguas de Lindoia, SP. |
Palavras-Chave: |
Disease; Resistance. |
Thesagro: |
Doença; Resistência; Soja. |
Thesaurus Nal: |
soybeans. |
Categoria do assunto: |
-- |
Marc: |
LEADER 00770naa a2200229 a 4500 001 1444838 005 2000-10-05 008 2000 bl uuuu u00u1 u #d 100 1 $aJUNG, R. 245 $aResistencia da cultivar FT-2 a mancha parda (Septoria glycines) avaliadas em geracoes F2 e F3 em soja. 260 $c2000 500 $aTrabalho apresentado no 46, Congresso Nacional de Genetica, 19 a 23 de setembro de 2000, Aguas de Lindoia, SP. 650 $asoybeans 650 $aDoença 650 $aResistência 650 $aSoja 653 $aDisease 653 $aResistance 700 1 $aTOLEDO, J. F. F. de 700 1 $aARIAS, C. A. A. 700 1 $aYORINORI, J. T. 773 $tGenetics and Molecular Biology, Ribeirao Preto$gv.23, n.3, p.547, Sept. 2000. Supplement.
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Registro original: |
Embrapa Soja (CNPSO) |
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Registro Completo
Biblioteca(s): |
Embrapa Milho e Sorgo. |
Data corrente: |
24/01/2020 |
Data da última atualização: |
24/01/2020 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
B - 2 |
Autoria: |
SOUZA, I. R. P. de; GUILHEN, J. H. S.; ANDRADE, C. de L. T. de; PINTO, M. de O.; LANA, U. G. de P.; PASTINA, M. M. |
Afiliação: |
ISABEL REGINA PRAZERES DE SOUZA, CNPMS; JOSÉ HENRIQUE SOLER GUILHEN, Universidade Federal do Espírito Santo; CAMILO DE LELIS TEIXEIRA DE ANDRADE, CNPMS; MARCOS DE OLIVEIRA PINTO, CNPMS; UBIRACI GOMES DE PAULA LANA, CNPMS; MARIA MARTA PASTINA, CNPMS. |
Título: |
Major effect qtl on chromosome 3 conferring maize resistance to Sugarcane mosaic virus. |
Ano de publicação: |
2019 |
Fonte/Imprenta: |
Revista Brasileira de Milho e Sorgo, v. 18, n. 3, p. 322-339, 2019. |
Idioma: |
Inglês |
Conteúdo: |
The Sugarcane mosaic virus (SCMV), a maize pathogen epidemic worldwide, is the causal agent of common mosaic, one of the most important viral diseases in Brazil. In this study, we mapped and characterized quantitative trait loci (QTL) conferring resistance to SCMV in a maize population consisting of 127 F2:3 families from the cross between two Brazilian maize inbred lines, L18 (resistant) × L19 (susceptible). Field trials were carried out in two years to evaluate the F2:3 families according to a resistance score after artificial inoculation. QTLs were detected via composite interval mapping, using a linkage map based on 82 SSRs, 3 CAPS and 296 SNPs. The heritability ranged from 73.68 to 95.16% and SCMV resistance QTLs were consistently identified on chromosomes 1 and 3, showing minor and major effects, respectively. The major QTL on chromosome 3 explained a large proportion of the genetic variance, being 50 and 70% in year 1 and 2, respectively, while the minor QTL on chromosome 1 explained 11 and 8% in year 1 and 2, respectively. The SNP marker co-localized with the major QTL peak on chromosome 3 and its right flanking marker are positioned inside the predicted gene GRMZM2G122443 encoding a glucosidase II, and the left flanking marker inside the GRMZM2G140537 that encodes a protein tyrosine kinase. Moreover, within this QTL region there are also the GRMZM2G160902 and GRMZM2G122481 predicted genes, encoding a bZIP transcription factor and a cytochrome C oxidase, respectively. The colocalization with this major effect QTL suggests a putative involvement of these candidate genes with maize responses to SCMV resistance, but further functional studies are required for such validation. Our results provide resistance source and genomic target for marker-assisted breeding aiming the development of maize resistant cultivars to SCMV. MenosThe Sugarcane mosaic virus (SCMV), a maize pathogen epidemic worldwide, is the causal agent of common mosaic, one of the most important viral diseases in Brazil. In this study, we mapped and characterized quantitative trait loci (QTL) conferring resistance to SCMV in a maize population consisting of 127 F2:3 families from the cross between two Brazilian maize inbred lines, L18 (resistant) × L19 (susceptible). Field trials were carried out in two years to evaluate the F2:3 families according to a resistance score after artificial inoculation. QTLs were detected via composite interval mapping, using a linkage map based on 82 SSRs, 3 CAPS and 296 SNPs. The heritability ranged from 73.68 to 95.16% and SCMV resistance QTLs were consistently identified on chromosomes 1 and 3, showing minor and major effects, respectively. The major QTL on chromosome 3 explained a large proportion of the genetic variance, being 50 and 70% in year 1 and 2, respectively, while the minor QTL on chromosome 1 explained 11 and 8% in year 1 and 2, respectively. The SNP marker co-localized with the major QTL peak on chromosome 3 and its right flanking marker are positioned inside the predicted gene GRMZM2G122443 encoding a glucosidase II, and the left flanking marker inside the GRMZM2G140537 that encodes a protein tyrosine kinase. Moreover, within this QTL region there are also the GRMZM2G160902 and GRMZM2G122481 predicted genes, encoding a bZIP transcription factor and a cytochrome C oxidase, respectively... Mostrar Tudo |
Thesagro: |
Gene; Linhagem; Marcador Genético; Zea Mays. |
Categoria do assunto: |
G Melhoramento Genético |
URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/209549/1/Major-effect.pdf
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Marc: |
LEADER 02540naa a2200229 a 4500 001 2119299 005 2020-01-24 008 2019 bl uuuu u00u1 u #d 100 1 $aSOUZA, I. R. P. de 245 $aMajor effect qtl on chromosome 3 conferring maize resistance to Sugarcane mosaic virus.$h[electronic resource] 260 $c2019 520 $aThe Sugarcane mosaic virus (SCMV), a maize pathogen epidemic worldwide, is the causal agent of common mosaic, one of the most important viral diseases in Brazil. In this study, we mapped and characterized quantitative trait loci (QTL) conferring resistance to SCMV in a maize population consisting of 127 F2:3 families from the cross between two Brazilian maize inbred lines, L18 (resistant) × L19 (susceptible). Field trials were carried out in two years to evaluate the F2:3 families according to a resistance score after artificial inoculation. QTLs were detected via composite interval mapping, using a linkage map based on 82 SSRs, 3 CAPS and 296 SNPs. The heritability ranged from 73.68 to 95.16% and SCMV resistance QTLs were consistently identified on chromosomes 1 and 3, showing minor and major effects, respectively. The major QTL on chromosome 3 explained a large proportion of the genetic variance, being 50 and 70% in year 1 and 2, respectively, while the minor QTL on chromosome 1 explained 11 and 8% in year 1 and 2, respectively. The SNP marker co-localized with the major QTL peak on chromosome 3 and its right flanking marker are positioned inside the predicted gene GRMZM2G122443 encoding a glucosidase II, and the left flanking marker inside the GRMZM2G140537 that encodes a protein tyrosine kinase. Moreover, within this QTL region there are also the GRMZM2G160902 and GRMZM2G122481 predicted genes, encoding a bZIP transcription factor and a cytochrome C oxidase, respectively. The colocalization with this major effect QTL suggests a putative involvement of these candidate genes with maize responses to SCMV resistance, but further functional studies are required for such validation. Our results provide resistance source and genomic target for marker-assisted breeding aiming the development of maize resistant cultivars to SCMV. 650 $aGene 650 $aLinhagem 650 $aMarcador Genético 650 $aZea Mays 700 1 $aGUILHEN, J. H. S. 700 1 $aANDRADE, C. de L. T. de 700 1 $aPINTO, M. de O. 700 1 $aLANA, U. G. de P. 700 1 $aPASTINA, M. M. 773 $tRevista Brasileira de Milho e Sorgo$gv. 18, n. 3, p. 322-339, 2019.
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