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Registros recuperados : 53 | |
21. | | DAVILA, A. M. R.; SOUZA, S. S.; CAMPOS, C.; SILVA, R. A. M. S. The seroprevalence of equine Trypanosomosis in the Pantanal. Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.94, n.2, p.199-202, mar./apr., 1999. Biblioteca(s): Embrapa Pantanal. |
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25. | | SILVA, R. A. M. S.; VICTORIO, A. M.; RAMIREZ, L.; DAVILA, A. M. R.; TRAJANO, V.; JANSEN, A. M. Effects of Trypanosoma evansi on the blood chemistry and hematology of coatis (Nasua nasua) naturally infected in the Pantanal, Brazil. Memorias do Instituto Oswaldo Cruz, v.92, p.122, Nov. 1997. Supplement 1. Biblioteca(s): Embrapa Pantanal. |
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26. | | SILVA, R. A. M. S.; RAMIREZ, L.; SOUZA, S. S.; ORTIZ, A. G.; PEREIRA, S. R.; DAVILA, A. M. R. Hematology of natural bovine trypanosomosis in the Brazilian Pantanal and Bolivian wetlands. Veterinary Parasitology, Amsterdam, v.85, n.1, p.87-93, 1999. Biblioteca(s): Embrapa Pantanal. |
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27. | | SILVA, R. A. M. S.; DAVILA, A. M. R.; PEREIRA, M. E. B.; RAMIREZ, L.; SANCHES, W.; GUTIERREZ, J. L. Bovine trypanosomiasis due to Trypanosoma vivax in the German Bush province, Bolivia. Memorias do Instituto Oswaldo Cruz, v.92, p.124, Nov. 1997. Supplement 1. Biblioteca(s): Embrapa Pantanal. |
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28. | | SILVA, R. A. M. S.; HERRERA, H. M.; DOMINGOS, L. B. da S.; XIMENES, F. A.; DAVILA, A. M. R. Pathogenesis of Trypanosoma evansi infection in dogs and horses: hematological and clinical aspects. Ciencia Rural, Santa Maria, v.25, n.2, p.233-238, maio/ago., 1995. Biblioteca(s): Embrapa Pantanal. |
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32. | | RAMIREZ, L.; DAVILA, A. M. R.; VICTORIO, A. M.; SILVA, R. A. M. S.; TRAJANO, V.; JANSEN, A. M. Measurements of Trypanosoma evansi from the Pantanal. Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.92, n.4, p.483-484, jul./ago., 1997. Biblioteca(s): Embrapa Pantanal. |
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36. | | SILVA, R. A. M. S.; RAMIREZ, L.; DAVILA, A. M. R.; FERREIRA, M. J. da S.; SAHIB, C. A. The first record of outbreak of bovine trypanosomosis by Trypanosoma vivax in Mato Grosso do Sul State, Brazil. ARS Veterinaria, v.14, n.2, p.129-133, 1998. Biblioteca(s): Embrapa Pantanal. |
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37. | | SILVA, R. A. M. S.; RAMIREZ, L.; DAVILA, A. M. R.; FERREIRA, M. J. da S.; SAHIB, C. A. The first record of outbreaks of bovine Trypanosomosis of Trypanosoma vivax in Mato Grosso do Sul State, Brazil. ARS Veterinária, v.14, n.2, p.129-133, 1998. Biblioteca(s): Embrapa Suínos e Aves. |
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39. | | SILVA, R. A. M. S.; SAHIB, C. A.; RUE, M. de la; HERRERA, H. M.; FERREIRA, M. J.; DAVILA, A. M. R. Coagulopathy due to Trypanosoma evansi acute infection in a dog. Arquivo Brasileiro de Medicina Veterinaria e Zootecnia, Belo Horizonte, v.48, n.4, p.485-489, ago. 1996. Biblioteca(s): Embrapa Pantanal. |
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40. | | RAMIREZ, L.; DAVILA, A. M. R.; VICTORIO, A. M.; SILVA, R. A. M. S.; VALQUIRIA, T.; JANSEN, A. M.; LEON, L. Biometrical and morphological differences in Trypanosoma evansi: primary isolates from wild and domestic animal of the Pantanal. Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.91, p.106, 1996. Suplemento. Biblioteca(s): Embrapa Pantanal. |
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Registros recuperados : 53 | |
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| Acesso ao texto completo restrito à biblioteca da Embrapa Pantanal. Para informações adicionais entre em contato com cpap.biblioteca@embrapa.br. |
Registro Completo
Biblioteca(s): |
Embrapa Pantanal. |
Data corrente: |
11/09/1997 |
Data da última atualização: |
11/09/1997 |
Autoria: |
SILVA, R. A. M. S.; RAMIREZ, L.; DAVILA, A. M. R.; PEREIRA, M. E. B.; HERRERA, H. M. |
Afiliação: |
EMBRAPA. Centro de Pesquisa Agropecuaria do Pantanal (Corumba, MS); UFMS. Centro Universitario de Corumba (Corumba, MS). |
Título: |
Infection in wistar rats after oral inoculation of Trypanosoma evansi. |
Ano de publicação: |
1995 |
Fonte/Imprenta: |
Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.90, p.298, 1995. Suplemento I. |
Idioma: |
Inglês |
Conteúdo: |
Trypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its length. The spleen of rats inoculated i.p. increased two and half times its weight and only aproximatelly once and half its length. The liver and lungs were intensively hyperemic and the kidneys were congested in all infected animals. Similar alterations has been reported in T. brucei infection in mice and rats and by T. evansi in dogs. The high infectivity of T. evansi to rats through oral route could means that the oral important route in a sylvatic cycle. MenosTrypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its len... Mostrar Tudo |
Thesaurus NAL: |
Pantanal; Trypanosoma evansi. |
Categoria do assunto: |
-- |
Marc: |
LEADER 02527naa a2200193 a 4500 001 1791636 005 1997-09-11 008 1995 bl uuuu u00u1 u #d 100 1 $aSILVA, R. A. M. S. 245 $aInfection in wistar rats after oral inoculation of Trypanosoma evansi. 260 $c1995 520 $aTrypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its length. The spleen of rats inoculated i.p. increased two and half times its weight and only aproximatelly once and half its length. The liver and lungs were intensively hyperemic and the kidneys were congested in all infected animals. Similar alterations has been reported in T. brucei infection in mice and rats and by T. evansi in dogs. The high infectivity of T. evansi to rats through oral route could means that the oral important route in a sylvatic cycle. 650 $aPantanal 650 $aTrypanosoma evansi 700 1 $aRAMIREZ, L. 700 1 $aDAVILA, A. M. R. 700 1 $aPEREIRA, M. E. B. 700 1 $aHERRERA, H. M. 773 $tMemorias do Instituto Oswaldo Cruz, Rio de Janeiro$gv.90, p.298, 1995. Suplemento I.
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