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| 18. |  | CHARCHAR, J. M.; EISENBACK, J. D.; CHARCHAR, M. J. D.; FONSECA, M. E. N. Meloidogyne phaseoli n. sp. (Nematoda: Meloidogynidae), a root-knot nematode parasitising bean in Brazil. Nematology, Leider, v. 10, n. 4, p. 525-538, 2008. Na publicação: Maria Esther N. F. BOITEUX| Biblioteca(s): Embrapa Hortaliças. |
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| 19. | | CHARCHAR, J. M.; EISENBACK, J. D.; CHARCHAR, M. J. D.; FONSECA, M. E. N. Meloidogyne pisi n. sp. (Nematoda: Meloidogynidae), a root-knot nematode parasitising pea in Brazil. Nematology, Leider, v. 10, n. 4, p. 429-493, 2008. Na publicação: Maria Esther N. F. BOITEUX| Biblioteca(s): Embrapa Hortaliças. |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Gado de Corte. Para informações adicionais entre em contato com cnpgc.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Gado de Corte. |
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Data corrente: |
10/07/1998 |
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Data da última atualização: |
10/07/1998 |
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Autoria: |
CHAKRABORTY, S.; PERROT, R.; CHARCHAR, M. J. D`A.; FERNANDES, C. D.; KELEMU, S. |
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Afiliação: |
CSIRO. Tropical Agriculture (Queensland, Australia). |
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Título: |
Biodiversity, epidemiology and virulence of Colletotrichum gloeosporioides. II. Genetic and pathogenic diversity in isolates of Colletotrichum gloeosporioides from eight species of Stylosanthes. |
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Ano de publicação: |
1997 |
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Fonte/Imprenta: |
Tropical Grasslands, Brisbane, v.31, n.5, p.393-401, Nov. 1997. |
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Idioma: |
Inglês |
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Notas: |
CNPGC. |
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Conteúdo: |
This work provides information on genetic and pathogenic variation in isolates of Collectotrichum gloeosporioides which cause anthracnose disease in many species of the tropical pasture legume, Stylosanthes. Isolates collected from species other than S. guianensis mainly from the centre of host-pathogen diversity in South America were studied using Random Amplified Polymorphic DNA (RAPD) markers and differential virulence on a set of host differentials. A phenetic analysis of 90 isolates using RAPD markers showed a wide genetic diversity in the pathogen population. The genetic variation in the Australian population was very limited compared with that at the centre of diversity. A similar situation was detected for pathogenic variation in 69 isolates from Brazil, many of which showed higher severity on Australian cultivars and accessions. These include accessions and cultivars which are highly resistant to the Australian races of the pathogen. However, pathogenic diversity in the Brazilian population of C. gloeosporioides was only partly characterised due to the limited number of host differentials used in this study. Attention needs to focus on the development and use of a more extensive range of host differentials. There was no strong relationship between genetic and virulence markers. Consequently, pathogenicity tests need to be conducted to answer the more practical questions such as the risk to current commercial cultivars from markers will damaging races. Use of molecular be useful to understand better the movement of pathogen populations between geographically isolated regions. Isolates used in this work have come mainly from regions where S. capitata and S. guianensis are endemic. Further isolate sampling from regions representing the native range of commercial species such as S. scabra and S. hamata will be necessary for a more comprehensive understating of this diversity. MenosThis work provides information on genetic and pathogenic variation in isolates of Collectotrichum gloeosporioides which cause anthracnose disease in many species of the tropical pasture legume, Stylosanthes. Isolates collected from species other than S. guianensis mainly from the centre of host-pathogen diversity in South America were studied using Random Amplified Polymorphic DNA (RAPD) markers and differential virulence on a set of host differentials. A phenetic analysis of 90 isolates using RAPD markers showed a wide genetic diversity in the pathogen population. The genetic variation in the Australian population was very limited compared with that at the centre of diversity. A similar situation was detected for pathogenic variation in 69 isolates from Brazil, many of which showed higher severity on Australian cultivars and accessions. These include accessions and cultivars which are highly resistant to the Australian races of the pathogen. However, pathogenic diversity in the Brazilian population of C. gloeosporioides was only partly characterised due to the limited number of host differentials used in this study. Attention needs to focus on the development and use of a more extensive range of host differentials. There was no strong relationship between genetic and virulence markers. Consequently, pathogenicity tests need to be conducted to answer the more practical questions such as the risk to current commercial cultivars from markers will damaging races. Use of molecul... Mostrar Tudo |
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Palavras-Chave: |
Anthracnosis; Brasil; Colletotricum gloeosporioides. |
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Thesagro: |
Antracnose; Epidemiologia; Estilosante; Marcador Genético; Patogenicidade; Stylosanthes Guianensis; Stylosanthes Macrocephala; Stylosanthes Scabra; Variação Genética. |
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Thesaurus NAL: |
Australia; Brazil; Colombia; epidemiology; genetic markers; genetic variation; pathogenicity; Stylosanthes capitata; Stylosanthes hamata. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 03220naa a2200433 a 4500
001 1317327
005 1998-07-10
008 1997 bl uuuu u00u1 u #d
100 1 $aCHAKRABORTY, S.
245 $aBiodiversity, epidemiology and virulence of Colletotrichum gloeosporioides. II. Genetic and pathogenic diversity in isolates of Colletotrichum gloeosporioides from eight species of Stylosanthes.
260 $c1997
500 $aCNPGC.
520 $aThis work provides information on genetic and pathogenic variation in isolates of Collectotrichum gloeosporioides which cause anthracnose disease in many species of the tropical pasture legume, Stylosanthes. Isolates collected from species other than S. guianensis mainly from the centre of host-pathogen diversity in South America were studied using Random Amplified Polymorphic DNA (RAPD) markers and differential virulence on a set of host differentials. A phenetic analysis of 90 isolates using RAPD markers showed a wide genetic diversity in the pathogen population. The genetic variation in the Australian population was very limited compared with that at the centre of diversity. A similar situation was detected for pathogenic variation in 69 isolates from Brazil, many of which showed higher severity on Australian cultivars and accessions. These include accessions and cultivars which are highly resistant to the Australian races of the pathogen. However, pathogenic diversity in the Brazilian population of C. gloeosporioides was only partly characterised due to the limited number of host differentials used in this study. Attention needs to focus on the development and use of a more extensive range of host differentials. There was no strong relationship between genetic and virulence markers. Consequently, pathogenicity tests need to be conducted to answer the more practical questions such as the risk to current commercial cultivars from markers will damaging races. Use of molecular be useful to understand better the movement of pathogen populations between geographically isolated regions. Isolates used in this work have come mainly from regions where S. capitata and S. guianensis are endemic. Further isolate sampling from regions representing the native range of commercial species such as S. scabra and S. hamata will be necessary for a more comprehensive understating of this diversity.
650 $aAustralia
650 $aBrazil
650 $aColombia
650 $aepidemiology
650 $agenetic markers
650 $agenetic variation
650 $apathogenicity
650 $aStylosanthes capitata
650 $aStylosanthes hamata
650 $aAntracnose
650 $aEpidemiologia
650 $aEstilosante
650 $aMarcador Genético
650 $aPatogenicidade
650 $aStylosanthes Guianensis
650 $aStylosanthes Macrocephala
650 $aStylosanthes Scabra
650 $aVariação Genética
653 $aAnthracnosis
653 $aBrasil
653 $aColletotricum gloeosporioides
700 1 $aPERROT, R.
700 1 $aCHARCHAR, M. J. D`A.
700 1 $aFERNANDES, C. D.
700 1 $aKELEMU, S.
773 $tTropical Grasslands, Brisbane$gv.31, n.5, p.393-401, Nov. 1997.
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