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Registros recuperados : 10 | |
1. | | NÉSPOLI, P. B.; DUARTE, M. D.; BEZERRA JÚNIOR, P. S.; DÖBEREINER, Jürgen; PEIXOTO, P. V. Aspectos clínico-patológicos da intoxicação experimental por selenito de sódio em eqüinos = Clinical and pathological aspects of experimental poisoning by sodium selenite in horses.. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 21, n. 3, p. 109-116, 2001. Biblioteca(s): Embrapa Agrobiologia. |
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2. | | RAYMUNDO, D. L.; BEZERRA JÚNIOR, P. S.; BANDARRA, P. M.; PEDROSO, P. M. O.; OLIVIERA, E. C.; PESCADOR, C. A.; DRIEMEIER, D. Spontaneous poisoning by larvae of Perreyia flavipes (Pergidae) in sheep. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 28, n. 1, p. 19-22, jan., 2008. Biblioteca(s): Embrapa Caprinos e Ovinos. |
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3. | | PEIXOTO, P. V.; OLIVEIRA, K. D.; FRANÇA, T. N.; DRIEMEIER, D.; DUARTE, M. D.; BEZERRA JUNIOR, P. S.; CERQUEIRA, V. D.; ARMIÉN, A. G. Experimental and iatrogenic poisoning by sodium selenite in pigs. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 37, n. 6, p. 561-569, jun. 2017. Título em inglês: Intoxicação iatrogênica e experimental por selenito de sódio em suínos. Biblioteca(s): Embrapa Unidades Centrais. |
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4. | | BEZERRA JUNIOR, P. S.; SANTOS, A. da S.; BANDARRA, P. M.; PEDROSO, P. M. O.; PAVARINI, S. P.; SPANAMBERG, A.; FERREIRO, L.; DRIEMEIER, D. Intoxicação experimental por Aspergillus clavatus em ovinos. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 29, n. 3, p. 205-210, mar., 2009. Biblioteca(s): Embrapa Caprinos e Ovinos. |
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5. | | TRUJILLO, J. G.; TEIXEIRA, M. A. S.; LIMA, A. H. A.; MONTÃO, D. P.; ROSS, T. B.; RIET-CORREA, G.; CERQUEIRA, V. D.; BEZERRA JÚNIOR, P. S. Intoxicação por etanol em bovinos alimentados com bagaço de malte acrescido de levedura de cerveja. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 38, n. 3, p. 382-386, março 2018 Título em inglês: Ethanol poisoning in cattle fed with malted barley waste with brewer?s yeast. Biblioteca(s): Embrapa Unidades Centrais. |
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6. | | REIS, A. S. B.; BRITO, M. F.; BEZERRA JUNIOR, P. S.; FONSECA JUNIOR, A. A.; UBIALI, D. G.; MOTA, R. A.; LEITE, R. C.; BARBOSA, J. D. Diagnóstico de paratuberculose por biópsia retal em búfalos. Pesquisa Veterinária Brasileira, Brasília, DF, v. 35, n. 10, p. 823-828, out. 2015. Biblioteca(s): Embrapa Unidades Centrais. |
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7. | | BRUST, L. A. C.; ARAGÃO, A. P.; BEZERRA JUNIOR, P. S.; GALVÃO, A.; FRANÇA, T. N.; GRAÇA, F. A. S.; PEIXOTO, P. V. Enfermidades em bovinos associadas ao consumo de resíduos de cervejaria. Pesquisa Veterinária Brasileira, Brasília, DF, v. 35, n. 12, p. 956-964, dez. 2015. Biblioteca(s): Embrapa Unidades Centrais. |
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8. | | COSTA, R. C.; AMBRÓSIO, N. A.; SOARES, B. A.; BEZERRA JÚNIOR, P. S.; BARÇANTE, T. A.; BARRIOS, P. R.; BARÇANTE, J. M. P. Pathological and parasitological aspects of the peacock (Pavo cristatus) infection by Tanaisia (Paratanaisia) bragai. Pesquisa Veterinária Brasileira, Rio de Janeiro v. 35, n. 5, p. 466-469, maio 2015. Biblioteca(s): Embrapa Unidades Centrais. |
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9. | | TEIXEIRA, M. A. S.; MACHADO, F. M. C.; SARMENTO, N. M. F. P.; OLIVEIRA JÚNIOR, C. A.; RIET-CORREA, G.; CERQUEIRA, V. D.; FRANÇA, T. do N.; BEZERRA JÚNIOR, P. S. Aspectos patológicos e imuno-histoquímicos da polisserosite em búfalos (Bubalus bubalis). Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 38, n. 1, p. 59-64, janeiro 2018. Título em inglês: Histopathological and immunohistochemical study of the polyserositis in buffalo (Bubalus bubalis). Biblioteca(s): Embrapa Unidades Centrais. |
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10. | | PEREIRA, J. D. B.; CERQUEIRA, V. D.; BEZERRA JUNIOR, P. S.; BEZERRA, D. K. O.; ARAUJO, F. R.; DIAS, A. de C. L.; ARAÚJO, C. P.; RIET-CORREA, G. Diagnóstico histopatológico e molecular de lesões sugestivas de tuberculose em búfalos abatidos nos municípios de Macapá e Santana, estado do Amapá. Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 37, n. 11, p. 1198-1204, novembro 2017. Título em inglês: Histopathological and molecular diagnosis of lesions suggestive of tuberculosis in buffaloes slaughtered in the municipalities of Macapá and Santana, Amapá state, Brazil. Biblioteca(s): Embrapa Gado de Corte; Embrapa Unidades Centrais. |
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Registros recuperados : 10 | |
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Registro Completo
Biblioteca(s): |
Embrapa Unidades Centrais. |
Data corrente: |
19/09/2017 |
Data da última atualização: |
29/12/2017 |
Autoria: |
PEIXOTO, P. V.; OLIVEIRA, K. D.; FRANÇA, T. N.; DRIEMEIER, D.; DUARTE, M. D.; BEZERRA JUNIOR, P. S.; CERQUEIRA, V. D.; ARMIÉN, A. G. |
Afiliação: |
Paulo V. Peixoto, Departamento de Nutrição Animal e Pastagem/Instituto de Zootecnia/Universidade Federal Rural do Rio de Janeiro - UFRRJ; Krishna D. Oliveira, Programa de Pós-Graduação em Medicina Veterinária/Instituto de Veterinária - IV/UFRRJ; Ticiana N. França, Departamento de Epidemiologia e Saúde Pública - DESP/IV-UFRRJ; David Driemeier, Departamento de Patologia e Clínica Veterinária/Faculdade de Medicina Veterinária/Universidade Federal do Rio Grande do Sul - UFRGS; Marcos D. Duarte, Instituto de Medicina Veterinária/Universidade Federal do Pará - UFPA; Pedro S. Bezerra Jr, Instituto de Medicina Veterinária/Universidade Federal do Pará - UFPA; Valíria D. Cerqueira, Instituto de Medicina Veterinária/Universidade Federal do Pará - UFPA; Aníbal G. Armién, Minnesota Veterinary Diagnostic Laboratory/Department of Veterinary Population/College of Veterinary Medicine/University of Minnesota. |
Título: |
Experimental and iatrogenic poisoning by sodium selenite in pigs. |
Ano de publicação: |
2017 |
Fonte/Imprenta: |
Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 37, n. 6, p. 561-569, jun. 2017. |
Idioma: |
Português |
Notas: |
Título em inglês: Intoxicação iatrogênica e experimental por selenito de sódio em suínos. |
Conteúdo: |
Following a case of iatrogenic selenium poisoning in a young pig, an experimental study was carry out. Sodium selenite was orally and parenterally administered to 13 pigs that were subdivided into three groups (G1, G2 and G3). The animals in groups G1 and G3 received sodium selenite intramuscularly (IM), G1 received a comercial formula, and G3 received sodium selenite mixed with distilled water at different dosages, and those in group G2 were fed commercial sodium selenite. Acute and subacute poisoning was observed in both groups, although the onset of clinical signs was slower in group G2. Only one pig (in group G1) that had received the highest dose showed a peracute course. Apathy, anorexia, dyspnea, vomiting, muscular tremors, proprioceptive deficit, ataxia and paresis of the hind limbs progressing to the front limbs evolving to tetraplegia were observed. Postmortem findings differed whether the animals received the injected (G1 and G3) or oral (G2) sodium selenite. The liver was moderately atrophic in some animals of G2. Some of the animals in groups G1 and G3 presented with lung edema. One pig in G3 had yellowish-brown areas in the ventral horns of the cervical intumescences of the spinal cord. The most important histological changes were present in the ventral horns of the cervical and lumbar intumescences of the spinal cord. In one animal, changes were present in the brainstem and mesencephalon. The initial lesion was a perivascular and astrocyte edema that progressing to lysis and death of astrocytes and neurons. In the chronic stage of the lesions, there were extensive areas of liquefaction necrosis with perivascular lymphocytic and histiocytic infiltration and occasional eosinophils. It seems that disruption of the blood-brain barrier due to astrocyte edema is the most likely mechanism of CNS lesion. MenosFollowing a case of iatrogenic selenium poisoning in a young pig, an experimental study was carry out. Sodium selenite was orally and parenterally administered to 13 pigs that were subdivided into three groups (G1, G2 and G3). The animals in groups G1 and G3 received sodium selenite intramuscularly (IM), G1 received a comercial formula, and G3 received sodium selenite mixed with distilled water at different dosages, and those in group G2 were fed commercial sodium selenite. Acute and subacute poisoning was observed in both groups, although the onset of clinical signs was slower in group G2. Only one pig (in group G1) that had received the highest dose showed a peracute course. Apathy, anorexia, dyspnea, vomiting, muscular tremors, proprioceptive deficit, ataxia and paresis of the hind limbs progressing to the front limbs evolving to tetraplegia were observed. Postmortem findings differed whether the animals received the injected (G1 and G3) or oral (G2) sodium selenite. The liver was moderately atrophic in some animals of G2. Some of the animals in groups G1 and G3 presented with lung edema. One pig in G3 had yellowish-brown areas in the ventral horns of the cervical intumescences of the spinal cord. The most important histological changes were present in the ventral horns of the cervical and lumbar intumescences of the spinal cord. In one animal, changes were present in the brainstem and mesencephalon. The initial lesion was a perivascular and astrocyte edema that progressi... Mostrar Tudo |
Palavras-Chave: |
Focal symmetrical poliomyelomalacia; Patogênese; Selenium poisoning. |
Thesagro: |
Suíno. |
Thesaurus NAL: |
Pathogenesis; Sodium selenite; Swine. |
Categoria do assunto: |
-- |
URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/163973/1/Experimental-and-iatrogenic.pdf
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Marc: |
LEADER 02799naa a2200301 a 4500 001 2075882 005 2017-12-29 008 2017 bl uuuu u00u1 u #d 100 1 $aPEIXOTO, P. V. 245 $aExperimental and iatrogenic poisoning by sodium selenite in pigs.$h[electronic resource] 260 $c2017 500 $aTítulo em inglês: Intoxicação iatrogênica e experimental por selenito de sódio em suínos. 520 $aFollowing a case of iatrogenic selenium poisoning in a young pig, an experimental study was carry out. Sodium selenite was orally and parenterally administered to 13 pigs that were subdivided into three groups (G1, G2 and G3). The animals in groups G1 and G3 received sodium selenite intramuscularly (IM), G1 received a comercial formula, and G3 received sodium selenite mixed with distilled water at different dosages, and those in group G2 were fed commercial sodium selenite. Acute and subacute poisoning was observed in both groups, although the onset of clinical signs was slower in group G2. Only one pig (in group G1) that had received the highest dose showed a peracute course. Apathy, anorexia, dyspnea, vomiting, muscular tremors, proprioceptive deficit, ataxia and paresis of the hind limbs progressing to the front limbs evolving to tetraplegia were observed. Postmortem findings differed whether the animals received the injected (G1 and G3) or oral (G2) sodium selenite. The liver was moderately atrophic in some animals of G2. Some of the animals in groups G1 and G3 presented with lung edema. One pig in G3 had yellowish-brown areas in the ventral horns of the cervical intumescences of the spinal cord. The most important histological changes were present in the ventral horns of the cervical and lumbar intumescences of the spinal cord. In one animal, changes were present in the brainstem and mesencephalon. The initial lesion was a perivascular and astrocyte edema that progressing to lysis and death of astrocytes and neurons. In the chronic stage of the lesions, there were extensive areas of liquefaction necrosis with perivascular lymphocytic and histiocytic infiltration and occasional eosinophils. It seems that disruption of the blood-brain barrier due to astrocyte edema is the most likely mechanism of CNS lesion. 650 $aPathogenesis 650 $aSodium selenite 650 $aSwine 650 $aSuíno 653 $aFocal symmetrical poliomyelomalacia 653 $aPatogênese 653 $aSelenium poisoning 700 1 $aOLIVEIRA, K. D. 700 1 $aFRANÇA, T. N. 700 1 $aDRIEMEIER, D. 700 1 $aDUARTE, M. D. 700 1 $aBEZERRA JUNIOR, P. S. 700 1 $aCERQUEIRA, V. D. 700 1 $aARMIÉN, A. G. 773 $tPesquisa Veterinária Brasileira, Rio de Janeiro$gv. 37, n. 6, p. 561-569, jun. 2017.
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