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Registro Completo |
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Biblioteca(s): |
Embrapa Algodão. |
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Data corrente: |
21/12/2012 |
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Data da última atualização: |
21/12/2012 |
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Tipo da produção científica: |
Resumo em Anais de Congresso |
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Autoria: |
RAMOS, L. C.; NOBREGA, M. B. de M.; ANDRADE, F. P. de; SILVA FILHO, J. L. da; FREITAS, J. G.; BARBOSA, M. A. |
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Afiliação: |
Lamonier Chaves Ramos, Aluno de graduação em Ciências Biológicas pela UEPB; MARCIA BARRETO DE MEDEIROS NOBREGA, CNPA; FRANCISCO PEREIRA DE ANDRADE, CNPA; JOAO LUIS DA SILVA FILHO, CNPA; JALMI GUEDES FREITAS, CNPA; Mayara Aranha Barbosa, Aluna do curso de Licenciatura em Biologia na UVA. |
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Título: |
Divergência genética em seis genitores de mamoneira. |
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Ano de publicação: |
2012 |
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Fonte/Imprenta: |
CONGRESSO BRASILEIRO DE MAMONA, 5.; SIMPÓSIO INTERNACIONAL DE OLEAGINOSAS ENERGÉTICAS, 2.; FÓRUM CAPIXABA DE PINHÃO-MANSO, 1., 2012, Guarapari. Desafios e Oportunidades: anais. Campina Grande: Embrapa Algodão, 2012. |
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Páginas: |
p. 369 |
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Idioma: |
Português |
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Palavras-Chave: |
DESCRITORES MORFOLÓGICOS; MELHORAMENTO GENÉTICO; RICINUS COMMUNIS L. |
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Thesagro: |
Mamona. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/72749/1/MEL-047-P.173.pdf
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Marc: |
LEADER 00791nam a2200217 a 4500
001 1943457
005 2012-12-21
008 2012 bl uuuu u00u1 u #d
100 1 $aRAMOS, L. C.
245 $aDivergência genética em seis genitores de mamoneira.
260 $aCONGRESSO BRASILEIRO DE MAMONA, 5.; SIMPÓSIO INTERNACIONAL DE OLEAGINOSAS ENERGÉTICAS, 2.; FÓRUM CAPIXABA DE PINHÃO-MANSO, 1., 2012, Guarapari. Desafios e Oportunidades: anais. Campina Grande: Embrapa Algodão$c2012
300 $ap. 369
650 $aMamona
653 $aDESCRITORES MORFOLÓGICOS
653 $aMELHORAMENTO GENÉTICO
653 $aRICINUS COMMUNIS L
700 1 $aNOBREGA, M. B. de M.
700 1 $aANDRADE, F. P. de
700 1 $aSILVA FILHO, J. L. da
700 1 $aFREITAS, J. G.
700 1 $aBARBOSA, M. A.
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Registro original: |
Embrapa Algodão (CNPA) |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Florestas. Para informações adicionais entre em contato com cnpf.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Florestas. |
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Data corrente: |
14/08/2009 |
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Data da última atualização: |
03/08/2022 |
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Tipo da produção científica: |
Artigo em Periódico Indexado |
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Circulação/Nível: |
A - 2 |
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Autoria: |
BASTIANEL, M.; CRISTOFANI-YALI, M.; OLIVEIRA, A. C. de; ASTUA, J. de F.; GARCIA, A. A. F.; RESENDE, M. D. V. de; RODRIGUES, V.; MACHADO, M. A. |
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Afiliação: |
JULIANA DE FREITAS ASTUA, CNPMF; MARCOS DEON VILELA DE RESENDE, CNPF. |
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Título: |
Quantitative trait loci analysis of citrus leprosis resistance in an interspecific backcross family of (Citrus riticulata Blanco x C. sinensis L. Osbeck) x C. sinensis L. Osb. |
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Ano de publicação: |
2009 |
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Fonte/Imprenta: |
Euphytica, v. 169, n. 1, p. 101-111, set. 2009. |
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Idioma: |
Inglês |
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Conteúdo: |
Leprosis, caused by citrus leprosis virus
(CiLV) and transmitted by the tenuipalpid mite
Brevipalpus phoenicis, is one of the most important
viruses of citrus in the Americas. Sweet oranges
(Citrus sinensis L. Osb.) are highly susceptible to
CiLV, while mandarins (C. reticulata Blanco) and
some of their hybrids have higher tolerance or resistance
to this disease. The mechanisms involved in the
resistance and its inheritance are still largely unknown.
To study the quantitative trait loci (QTL; quantitative
trait loci) associated with the resistance to CiLV,
progeny analyses were established with 143 hybrid
individuals of ?Pêra? sweet orange (C. sinensis L. Osb.)
and ?Murcott? tangor (C. reticulata Blanco 9
C. sinensis L. Osb.) from controlled crossings. Disease
assessment of the hybrid individuals was conducted by
infesting the plants with viruliferous mites in the field.
The experiment consisted of a randomized completely
block design with ten replicates. The evaluated phenotypic
traits were incidence and severity of the
disease on leaves and branches, for a period of 3 years.
The MapQTLTM v.4.0 software was used for the
identification and location of possible QTL associated
with resistance to CiLV on a genetic map obtained
from 260 AFLP and 5 RAPD markers. Only consistent
QTLs from different phenotypic traits and years of
evaluation, with the critical LOD scores to determine
the presence or absence of each QTL calculated
through the randompermutation test, were considered.
AQTL was observed and had a significant effect on the
phenotypic variation, ranging from 79.4 to 84%
depending on which trait (incidence or severity) was
assessed. This suggests that few genes are involved in
the genetic resistance of citrus to CiLV. MenosLeprosis, caused by citrus leprosis virus
(CiLV) and transmitted by the tenuipalpid mite
Brevipalpus phoenicis, is one of the most important
viruses of citrus in the Americas. Sweet oranges
(Citrus sinensis L. Osb.) are highly susceptible to
CiLV, while mandarins (C. reticulata Blanco) and
some of their hybrids have higher tolerance or resistance
to this disease. The mechanisms involved in the
resistance and its inheritance are still largely unknown.
To study the quantitative trait loci (QTL; quantitative
trait loci) associated with the resistance to CiLV,
progeny analyses were established with 143 hybrid
individuals of ?Pêra? sweet orange (C. sinensis L. Osb.)
and ?Murcott? tangor (C. reticulata Blanco 9
C. sinensis L. Osb.) from controlled crossings. Disease
assessment of the hybrid individuals was conducted by
infesting the plants with viruliferous mites in the field.
The experiment consisted of a randomized completely
block design with ten replicates. The evaluated phenotypic
traits were incidence and severity of the
disease on leaves and branches, for a period of 3 years.
The MapQTLTM v.4.0 software was used for the
identification and location of possible QTL associated
with resistance to CiLV on a genetic map obtained
from 260 AFLP and 5 RAPD markers. Only consistent
QTLs from different phenotypic traits and years of
evaluation, with the critical LOD scores to determine
the presence or absence of each QTL calculated
through the randompermutation test, were considered.
... Mostrar Tudo |
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Thesagro: |
Doença; Laranja; Leprose; Vírus. |
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Thesaurus NAL: |
Citrus. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 02571naa a2200265 a 4500
001 1298758
005 2022-08-03
008 2009 bl uuuu u00u1 u #d
100 1 $aBASTIANEL, M.
245 $aQuantitative trait loci analysis of citrus leprosis resistance in an interspecific backcross family of (Citrus riticulata Blanco x C. sinensis L. Osbeck) x C. sinensis L. Osb.$h[electronic resource]
260 $c2009
520 $aLeprosis, caused by citrus leprosis virus (CiLV) and transmitted by the tenuipalpid mite Brevipalpus phoenicis, is one of the most important viruses of citrus in the Americas. Sweet oranges (Citrus sinensis L. Osb.) are highly susceptible to CiLV, while mandarins (C. reticulata Blanco) and some of their hybrids have higher tolerance or resistance to this disease. The mechanisms involved in the resistance and its inheritance are still largely unknown. To study the quantitative trait loci (QTL; quantitative trait loci) associated with the resistance to CiLV, progeny analyses were established with 143 hybrid individuals of ?Pêra? sweet orange (C. sinensis L. Osb.) and ?Murcott? tangor (C. reticulata Blanco 9 C. sinensis L. Osb.) from controlled crossings. Disease assessment of the hybrid individuals was conducted by infesting the plants with viruliferous mites in the field. The experiment consisted of a randomized completely block design with ten replicates. The evaluated phenotypic traits were incidence and severity of the disease on leaves and branches, for a period of 3 years. The MapQTLTM v.4.0 software was used for the identification and location of possible QTL associated with resistance to CiLV on a genetic map obtained from 260 AFLP and 5 RAPD markers. Only consistent QTLs from different phenotypic traits and years of evaluation, with the critical LOD scores to determine the presence or absence of each QTL calculated through the randompermutation test, were considered. AQTL was observed and had a significant effect on the phenotypic variation, ranging from 79.4 to 84% depending on which trait (incidence or severity) was assessed. This suggests that few genes are involved in the genetic resistance of citrus to CiLV.
650 $aCitrus
650 $aDoença
650 $aLaranja
650 $aLeprose
650 $aVírus
700 1 $aCRISTOFANI-YALI, M.
700 1 $aOLIVEIRA, A. C. de
700 1 $aASTUA, J. de F.
700 1 $aGARCIA, A. A. F.
700 1 $aRESENDE, M. D. V. de
700 1 $aRODRIGUES, V.
700 1 $aMACHADO, M. A.
773 $tEuphytica$gv. 169, n. 1, p. 101-111, set. 2009.
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