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Registro Completo |
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Biblioteca(s): |
Embrapa Mandioca e Fruticultura. |
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Data corrente: |
16/03/1994 |
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Data da última atualização: |
16/03/1994 |
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Autoria: |
BROWN, G. ELDON; LEE, H. S. |
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Afiliação: |
Scientific Research Department, Florida Department of Citrus Researchand Education Center, Lake Alfred, FL 33850. We thank M. Chambers for technical assistance. |
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Título: |
Interactions of ethylene with citrus stem-end rot caused by Diplodia natalensis. |
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Ano de publicação: |
1993 |
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Fonte/Imprenta: |
Phytopathology, v.83, n.11, p.1204-1208, nov.,1993 |
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ISSN: |
0031-949X |
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Idioma: |
Inglês |
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Conteúdo: |
Ethylene treatment of citrus fruit to improve rind color (degreening)significantly enhances the incidence of stem-end rot caused by Diplodianatalensis and stimulates disease development. In an effort. In an effort to identify the role of execessive exogenous ethylene in pathogenesis, tissue exposed to different ethylene and air atmospheres was taken from the base of contrus fruit adjacent to the abscission area at the point of fungal ingress and examined for the presence of fungal inhibitors and lignin. The major inhibitor, identified as scoparone (6,7-dimethoxycoumarin), accumulated most extensively in tissue taken from asymptomatic fruit receiving either high ethylene treatments (55 u1 L-1) followed by low inculum levels, or low ethylene treatments (2 u1 L- 1) followed by high inoculum levels.Accumulation of lignin, extracted from the same tissue and measured as lignothiogucolic acid, followed areponse pattern similar to that observed for scoparone. Evidence for some role of scoparone and lignin in fruit resistance was indicated by the correlation between enhanced accumulation of these inhibitors and the lack of decay. However, other factors affected by high ethylene play a role in disease development since levels of inoculum that induced little disease at low ethylene were capalble of causing decay at high ethylene. |
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Palavras-Chave: |
Postharvest pathology; Scoparone. |
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Thesaurus Nal: |
lignin. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 01837naa a2200181 a 4500
001 1632997
005 1994-03-16
008 1993 bl uuuu u00u1 u #d
022 $a0031-949X
100 1 $aBROWN, G. ELDON
245 $aInteractions of ethylene with citrus stem-end rot caused by Diplodia natalensis.
260 $c1993
520 $aEthylene treatment of citrus fruit to improve rind color (degreening)significantly enhances the incidence of stem-end rot caused by Diplodianatalensis and stimulates disease development. In an effort. In an effort to identify the role of execessive exogenous ethylene in pathogenesis, tissue exposed to different ethylene and air atmospheres was taken from the base of contrus fruit adjacent to the abscission area at the point of fungal ingress and examined for the presence of fungal inhibitors and lignin. The major inhibitor, identified as scoparone (6,7-dimethoxycoumarin), accumulated most extensively in tissue taken from asymptomatic fruit receiving either high ethylene treatments (55 u1 L-1) followed by low inculum levels, or low ethylene treatments (2 u1 L- 1) followed by high inoculum levels.Accumulation of lignin, extracted from the same tissue and measured as lignothiogucolic acid, followed areponse pattern similar to that observed for scoparone. Evidence for some role of scoparone and lignin in fruit resistance was indicated by the correlation between enhanced accumulation of these inhibitors and the lack of decay. However, other factors affected by high ethylene play a role in disease development since levels of inoculum that induced little disease at low ethylene were capalble of causing decay at high ethylene.
650 $alignin
653 $aPostharvest pathology
653 $aScoparone
700 1 $aLEE, H. S.
773 $tPhytopathology$gv.83, n.11, p.1204-1208, nov.,1993
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Registro original: |
Embrapa Mandioca e Fruticultura (CNPMF) |
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