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Registro Completo |
Biblioteca(s): |
Embrapa Caprinos e Ovinos. |
Data corrente: |
01/08/1992 |
Data da última atualização: |
06/06/2023 |
Autoria: |
CORK, L. C.; HADLOW, W. J.; GORHAM, J. R.; PIPER, R. C.; CRAWFORD, T. B. |
Título: |
Pathology of viral leukoencephalomyelitis of goats. |
Ano de publicação: |
1974 |
Fonte/Imprenta: |
Acta Neuropathologica, v. 29, n. 4, p. 281-292, 1974. |
DOI: |
10.1007/BF00685482. |
Idioma: |
Inglês |
Conteúdo: |
The morphologic changes of viral leukoencephalomyelitis of goats (VLG), an afebrile paralytic disease of 2--4 month old kids, were studied in 13 naturally infected goats: 11 during the first 5--22 days of clinical disease and two that survived 8 months and 3 years after onset of clinical signs. Lesions in the early clinical phase of the disease included interstitial pneumonia and widely disseminated myelinoclastic perivascular lesions in the brain and spinal cord. In the central nervous system, hypertrophied and hyperplastic reticulin fibers of the vascular sheath encompassed an inflammatory cell infiltrate composed of lymphocytes, plasma cells, and macrophages. Occasionally, malacia occurred, and several animals had transverse myelitis. Neuronal necrosis and neuronophagia were not features of the disease. Lesions were most frequent in the subpia and subependyma. Pulmonary lesions were not present in goats that survived the initial clinical phase of the disease, but myelin had disappeared from large areas of the spinal cord and from small loci in the brain. Nodular accumulations of mononuclear cells and small perivascular cuffs were also present in the central nervous system. VLG resembles visna in the topographic distribution and myelinoclastic nature of its lesions. Like visna, it bears some resemblance to post-infectious encephalitis of man. |
Palavras-Chave: |
Demyelinating diseases; Doenca infecciosa; Encephalomyelitis; Virus diseases. |
Thesagro: |
Caprino; Doença; Encefalite; Sistema Nervoso. |
Thesaurus Nal: |
Animal disease models; Animal pathology; Brain; Disease transmission; Goat diseases; Goats; Lymphocytes; Plasma cells; Spinal cord. |
Categoria do assunto: |
-- |
Marc: |
LEADER 02389naa a2200385 a 4500 001 1522847 005 2023-06-06 008 1974 bl uuuu u00u1 u #d 024 7 $a10.1007/BF00685482.$2DOI 100 1 $aCORK, L. C. 245 $aPathology of viral leukoencephalomyelitis of goats.$h[electronic resource] 260 $c1974 520 $aThe morphologic changes of viral leukoencephalomyelitis of goats (VLG), an afebrile paralytic disease of 2--4 month old kids, were studied in 13 naturally infected goats: 11 during the first 5--22 days of clinical disease and two that survived 8 months and 3 years after onset of clinical signs. Lesions in the early clinical phase of the disease included interstitial pneumonia and widely disseminated myelinoclastic perivascular lesions in the brain and spinal cord. In the central nervous system, hypertrophied and hyperplastic reticulin fibers of the vascular sheath encompassed an inflammatory cell infiltrate composed of lymphocytes, plasma cells, and macrophages. Occasionally, malacia occurred, and several animals had transverse myelitis. Neuronal necrosis and neuronophagia were not features of the disease. Lesions were most frequent in the subpia and subependyma. Pulmonary lesions were not present in goats that survived the initial clinical phase of the disease, but myelin had disappeared from large areas of the spinal cord and from small loci in the brain. Nodular accumulations of mononuclear cells and small perivascular cuffs were also present in the central nervous system. VLG resembles visna in the topographic distribution and myelinoclastic nature of its lesions. Like visna, it bears some resemblance to post-infectious encephalitis of man. 650 $aAnimal disease models 650 $aAnimal pathology 650 $aBrain 650 $aDisease transmission 650 $aGoat diseases 650 $aGoats 650 $aLymphocytes 650 $aPlasma cells 650 $aSpinal cord 650 $aCaprino 650 $aDoença 650 $aEncefalite 650 $aSistema Nervoso 653 $aDemyelinating diseases 653 $aDoenca infecciosa 653 $aEncephalomyelitis 653 $aVirus diseases 700 1 $aHADLOW, W. J. 700 1 $aGORHAM, J. R. 700 1 $aPIPER, R. C. 700 1 $aCRAWFORD, T. B. 773 $tActa Neuropathologica$gv. 29, n. 4, p. 281-292, 1974.
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