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Registro Completo |
Biblioteca(s): |
Embrapa Arroz e Feijão. |
Data corrente: |
29/04/1994 |
Data da última atualização: |
17/08/2006 |
Autoria: |
LOPES, A. de M.; CRUZ, E. de S.; KASS, D. L. |
Título: |
Resposta do arroz apura, a adubação NPK, sob regime de irrigação natural: várzea do Rio Caeté-Município de Bragança-Pará. |
Ano de publicação: |
1973 |
Fonte/Imprenta: |
Belém: IPEAN, 1973. |
Páginas: |
7 p. |
Série: |
(IPEAN. Comunicado Técnico, 14). |
Idioma: |
Português |
Palavras-Chave: |
Apura; Cultivar; Fertilização; Pará. |
Thesagro: |
Adubação; Arroz; Irrigação; Npk; Oryza Sativa; Várzea. |
Categoria do assunto: |
-- |
Marc: |
LEADER 00717nam a2200265 a 4500 001 1195342 005 2006-08-17 008 1973 bl uuuu u0uu1 u #d 100 1 $aLOPES, A. de M. 245 $aResposta do arroz apura, a adubação NPK, sob regime de irrigação natural$bvárzea do Rio Caeté-Município de Bragança-Pará. 260 $aBelém: IPEAN$c1973 300 $a7 p. 490 $a(IPEAN. Comunicado Técnico, 14). 650 $aAdubação 650 $aArroz 650 $aIrrigação 650 $aNpk 650 $aOryza Sativa 650 $aVárzea 653 $aApura 653 $aCultivar 653 $aFertilização 653 $aPará 700 1 $aCRUZ, E. de S. 700 1 $aKASS, D. L.
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Registro original: |
Embrapa Arroz e Feijão (CNPAF) |
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| Acesso ao texto completo restrito à biblioteca da Embrapa Agroindústria de Alimentos. Para informações adicionais entre em contato com ctaa.biblioteca@embrapa.br. |
Registro Completo
Biblioteca(s): |
Embrapa Agroindústria de Alimentos. |
Data corrente: |
02/12/2016 |
Data da última atualização: |
25/09/2017 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
A - 1 |
Autoria: |
TONUCCI, L. B.; SANTOS, K. M. O. dos; OLIVEIRA, L. L. de; RIBEIRO, S. M. R.; MARTINO, H. S. D. |
Afiliação: |
Livia Bordalo Tonucci, INTA; KARINA MARIA OLBRICH DOS SANTOS, CTAA; Leandro Licursi de Oliveira, UFV; Sonia Machado Rocha Ribeiro, UFV; Hercia Stampini Duarte Martino, UFV. |
Título: |
Clinical application of probiotics in type 2 diabetes mellitus: a randomized, double-blind, placebo-controlled study. |
Ano de publicação: |
2017 |
Fonte/Imprenta: |
Clinical Nutrition, v. 36, n. 1, p. 85-92, 2017. |
DOI: |
10.1016/j.clnu.2015.11.011. |
Idioma: |
Inglês |
Conteúdo: |
Background & aims: Type 2 diabetes has been associated with dysbiosis and one of the possible routes to restore a healthy gut microbiota is by the regular ingestion of probiotics. We aimed to investigate the effects of probiotics on glycemic control, lipid profile, inflammation, oxidative stress and short chain fatty acids in T2D. Methods: In a double-blind, randomized, placebo-controlled trial, 50 volunteers consumed daily 120 g/d of fermented milk for 6 wk. Participants were assigned into two groups: probiotic group, consuming fermented milk containing Lactobacillus acidophilus La-5 and Bifidobacterium animalis subsp lactis BB-12 (109 colony-forming units/d, each) and control group, consuming conventional fermented milk. Anthropometric measurements, body composition, fasting blood and faecal samples were taken at baseline and after 6 wk. Results: 45 subjects out of 50 (90%) completed follow-up. After 6 wk, there was a significant decrease in fructosamine levels (9.91 mmol/L; p ¼ 0.04) and hemoglobin A1c tended to be lower (0.67%; p ¼ 0.06) in probiotic group. TNF-a and resistin were significantly reduced in probiotic and control groups (1.5 and 1.3 pg/mL, .1 and 2.8 ng/mL, respectively), while IL-10 was significantly reduced ( 0.65 pg/mL; p < 0.001) only in the control group. Fecal acetic acid was increased in both groups (0.58 and 0.59% in probiotic and control groups, respectively; p < 0.01). There was a significant difference between groups concerning mean changes of HbA1c (þ0.31 for control group vs 0.65 for probiotic group; p ¼ 0.02), total cholesterol (þ0.55 for control group vs 0.15 for probiotic group; p ¼ 0.04) and LDL-cholesterol (þ0.36 for control group vs 0.20 for probiotic group p ¼ 0.03). Conclusions: Probiotic consumption improved the glycemic control in T2D subjects, however, the intake of fermented milk seems to be involved with others metabolic changes, such as decrease in inflammatory cytokines (TNF-a and resistin) and increase in the acetic acid. MenosBackground & aims: Type 2 diabetes has been associated with dysbiosis and one of the possible routes to restore a healthy gut microbiota is by the regular ingestion of probiotics. We aimed to investigate the effects of probiotics on glycemic control, lipid profile, inflammation, oxidative stress and short chain fatty acids in T2D. Methods: In a double-blind, randomized, placebo-controlled trial, 50 volunteers consumed daily 120 g/d of fermented milk for 6 wk. Participants were assigned into two groups: probiotic group, consuming fermented milk containing Lactobacillus acidophilus La-5 and Bifidobacterium animalis subsp lactis BB-12 (109 colony-forming units/d, each) and control group, consuming conventional fermented milk. Anthropometric measurements, body composition, fasting blood and faecal samples were taken at baseline and after 6 wk. Results: 45 subjects out of 50 (90%) completed follow-up. After 6 wk, there was a significant decrease in fructosamine levels (9.91 mmol/L; p ¼ 0.04) and hemoglobin A1c tended to be lower (0.67%; p ¼ 0.06) in probiotic group. TNF-a and resistin were significantly reduced in probiotic and control groups (1.5 and 1.3 pg/mL, .1 and 2.8 ng/mL, respectively), while IL-10 was significantly reduced ( 0.65 pg/mL; p < 0.001) only in the control group. Fecal acetic acid was increased in both groups (0.58 and 0.59% in probiotic and control groups, respectively; p < 0.01). There was a significant difference between groups concerning mean changes of Hb... Mostrar Tudo |
Palavras-Chave: |
Gut microbiota; Oxidative; Type 2 diabetes. |
Thesagro: |
Stress. |
Thesaurus NAL: |
inflammation; probiotics. |
Categoria do assunto: |
X Pesquisa, Tecnologia e Engenharia |
Marc: |
LEADER 02784naa a2200253 a 4500 001 2057801 005 2017-09-25 008 2017 bl uuuu u00u1 u #d 024 7 $a10.1016/j.clnu.2015.11.011.$2DOI 100 1 $aTONUCCI, L. B. 245 $aClinical application of probiotics in type 2 diabetes mellitus$ba randomized, double-blind, placebo-controlled study.$h[electronic resource] 260 $c2017 520 $aBackground & aims: Type 2 diabetes has been associated with dysbiosis and one of the possible routes to restore a healthy gut microbiota is by the regular ingestion of probiotics. We aimed to investigate the effects of probiotics on glycemic control, lipid profile, inflammation, oxidative stress and short chain fatty acids in T2D. Methods: In a double-blind, randomized, placebo-controlled trial, 50 volunteers consumed daily 120 g/d of fermented milk for 6 wk. Participants were assigned into two groups: probiotic group, consuming fermented milk containing Lactobacillus acidophilus La-5 and Bifidobacterium animalis subsp lactis BB-12 (109 colony-forming units/d, each) and control group, consuming conventional fermented milk. Anthropometric measurements, body composition, fasting blood and faecal samples were taken at baseline and after 6 wk. Results: 45 subjects out of 50 (90%) completed follow-up. After 6 wk, there was a significant decrease in fructosamine levels (9.91 mmol/L; p ¼ 0.04) and hemoglobin A1c tended to be lower (0.67%; p ¼ 0.06) in probiotic group. TNF-a and resistin were significantly reduced in probiotic and control groups (1.5 and 1.3 pg/mL, .1 and 2.8 ng/mL, respectively), while IL-10 was significantly reduced ( 0.65 pg/mL; p < 0.001) only in the control group. Fecal acetic acid was increased in both groups (0.58 and 0.59% in probiotic and control groups, respectively; p < 0.01). There was a significant difference between groups concerning mean changes of HbA1c (þ0.31 for control group vs 0.65 for probiotic group; p ¼ 0.02), total cholesterol (þ0.55 for control group vs 0.15 for probiotic group; p ¼ 0.04) and LDL-cholesterol (þ0.36 for control group vs 0.20 for probiotic group p ¼ 0.03). Conclusions: Probiotic consumption improved the glycemic control in T2D subjects, however, the intake of fermented milk seems to be involved with others metabolic changes, such as decrease in inflammatory cytokines (TNF-a and resistin) and increase in the acetic acid. 650 $ainflammation 650 $aprobiotics 650 $aStress 653 $aGut microbiota 653 $aOxidative 653 $aType 2 diabetes 700 1 $aSANTOS, K. M. O. dos 700 1 $aOLIVEIRA, L. L. de 700 1 $aRIBEIRO, S. M. R. 700 1 $aMARTINO, H. S. D. 773 $tClinical Nutrition$gv. 36, n. 1, p. 85-92, 2017.
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