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Registro Completo |
Biblioteca(s): |
Embrapa Caprinos e Ovinos. |
Data corrente: |
06/09/2016 |
Data da última atualização: |
03/10/2016 |
Autoria: |
LANGLANDS, J. P.; BOWLES, J. E.; DONALD, G. E.; SMITH, A. J.; PAULL, D. R.; HOLMES, P. R. |
Título: |
Copper oxide particles for grazing sheep. |
Ano de publicação: |
1983 |
Fonte/Imprenta: |
Australian Journal of Agricultural Research, v. 34, n. 6, p. 751-765, 1983. |
DOI: |
10.1071/AR9830751 |
Idioma: |
Inglês |
Conteúdo: |
Abstract: In a series of experiments aimed at evaluating copper oxide as a supplement, grazing sheep were dosed with varying quantities of copper oxide particles up to 64 g, and the toxicity, the rate of particle excretion, and copper storage in the liver and other tissues were recorded. The toxicities (LD50) of copper oxide particles were 1.17 and 1.80 g/kg liveweight for two groups of grazing adult fine-wool Merino sheep. Death usually occurred 88-96 days after the oral administration of the particles; mean hepatic copper concentrations of sheep dying from copper toxicity were 4122-4308 mg/kg DM. The rate of faecal copper excretion of copper-supplemented sheep, expressed as a percentage of the dose, was less when 50 g of particles were given than when the dose was 5 or 10 g, but excretion patterns were variable. The quantity of hepatic copper stored per g of copper oxide given declined as the dose increased from 0 to 32 g, but increased again at higher doses. Hepatic copper concentration reached a maximum 2-3 months after dosing and the rate of decline was positively related to dose rate; thus, large doses of copper are unlikely to extend the period of elevated copper status. Large doses also increased whole blood copper concentrations and elevated plasma aspartate transaminase activities; this was taken to indicate copper poisoning. Tissue copper concentrations from sheep given up to 64 g particles are reported; liver was the most sensitive to copper treatment. Copper contents of the lung and kidney also responded to copper therapy, but carcass components such as leg, shoulder and muscle did not. Weaned lambs given 2 g of particles (c. 0.13 g/kg liveweight) grew significantly faster than unsupplemented lambs. This dose rate was approximately one-seventh of that predicted to cause 5% mortality, and it is concluded that, at appropriate dose rates, this method of supplementation did not increase mortality or cause excessive increases in tissue copper concentrations, and could increase growth rate. MenosAbstract: In a series of experiments aimed at evaluating copper oxide as a supplement, grazing sheep were dosed with varying quantities of copper oxide particles up to 64 g, and the toxicity, the rate of particle excretion, and copper storage in the liver and other tissues were recorded. The toxicities (LD50) of copper oxide particles were 1.17 and 1.80 g/kg liveweight for two groups of grazing adult fine-wool Merino sheep. Death usually occurred 88-96 days after the oral administration of the particles; mean hepatic copper concentrations of sheep dying from copper toxicity were 4122-4308 mg/kg DM. The rate of faecal copper excretion of copper-supplemented sheep, expressed as a percentage of the dose, was less when 50 g of particles were given than when the dose was 5 or 10 g, but excretion patterns were variable. The quantity of hepatic copper stored per g of copper oxide given declined as the dose increased from 0 to 32 g, but increased again at higher doses. Hepatic copper concentration reached a maximum 2-3 months after dosing and the rate of decline was positively related to dose rate; thus, large doses of copper are unlikely to extend the period of elevated copper status. Large doses also increased whole blood copper concentrations and elevated plasma aspartate transaminase activities; this was taken to indicate copper poisoning. Tissue copper concentrations from sheep given up to 64 g particles are reported; liver was the most sensitive to copper treatment. Copper con... Mostrar Tudo |
Palavras-Chave: |
Copper oxide; Óxido de cobre. |
Thesagro: |
Nutrição animal; Ovino; Pastejo; Suplemento mineral. |
Thesaurus Nal: |
Sheep. |
Categoria do assunto: |
K Ciência Florestal e Produtos de Origem Vegetal |
Marc: |
LEADER 02760naa a2200277 a 4500 001 2052478 005 2016-10-03 008 1983 bl uuuu u00u1 u #d 024 7 $a10.1071/AR9830751$2DOI 100 1 $aLANGLANDS, J. P. 245 $aCopper oxide particles for grazing sheep. 260 $c1983 520 $aAbstract: In a series of experiments aimed at evaluating copper oxide as a supplement, grazing sheep were dosed with varying quantities of copper oxide particles up to 64 g, and the toxicity, the rate of particle excretion, and copper storage in the liver and other tissues were recorded. The toxicities (LD50) of copper oxide particles were 1.17 and 1.80 g/kg liveweight for two groups of grazing adult fine-wool Merino sheep. Death usually occurred 88-96 days after the oral administration of the particles; mean hepatic copper concentrations of sheep dying from copper toxicity were 4122-4308 mg/kg DM. The rate of faecal copper excretion of copper-supplemented sheep, expressed as a percentage of the dose, was less when 50 g of particles were given than when the dose was 5 or 10 g, but excretion patterns were variable. The quantity of hepatic copper stored per g of copper oxide given declined as the dose increased from 0 to 32 g, but increased again at higher doses. Hepatic copper concentration reached a maximum 2-3 months after dosing and the rate of decline was positively related to dose rate; thus, large doses of copper are unlikely to extend the period of elevated copper status. Large doses also increased whole blood copper concentrations and elevated plasma aspartate transaminase activities; this was taken to indicate copper poisoning. Tissue copper concentrations from sheep given up to 64 g particles are reported; liver was the most sensitive to copper treatment. Copper contents of the lung and kidney also responded to copper therapy, but carcass components such as leg, shoulder and muscle did not. Weaned lambs given 2 g of particles (c. 0.13 g/kg liveweight) grew significantly faster than unsupplemented lambs. This dose rate was approximately one-seventh of that predicted to cause 5% mortality, and it is concluded that, at appropriate dose rates, this method of supplementation did not increase mortality or cause excessive increases in tissue copper concentrations, and could increase growth rate. 650 $aSheep 650 $aNutrição animal 650 $aOvino 650 $aPastejo 650 $aSuplemento mineral 653 $aCopper oxide 653 $aÓxido de cobre 700 1 $aBOWLES, J. E. 700 1 $aDONALD, G. E. 700 1 $aSMITH, A. J. 700 1 $aPAULL, D. R. 700 1 $aHOLMES, P. R. 773 $tAustralian Journal of Agricultural Research$gv. 34, n. 6, p. 751-765, 1983.
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Registro original: |
Embrapa Caprinos e Ovinos (CNPC) |
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Registro Completo
Biblioteca(s): |
Embrapa Hortaliças. |
Data corrente: |
13/11/2002 |
Data da última atualização: |
13/11/2002 |
Autoria: |
MAROUELLI, W. A.; SILVA, W. L. C.; MORETTI, C. L. |
Afiliação: |
Embrapa Hortaliças, Brasília, DF. |
Título: |
Desenvolvimento de plantas, produção e qualidade de bulbos de alho sob condições de deficiência de água no solo. |
Ano de publicação: |
2002 |
Fonte/Imprenta: |
Horticultura Brasileira, Brasília, DF, v. 20, n. 3, p. 470-473, set. 2002. |
Idioma: |
Português |
Palavras-Chave: |
Brasil; Brasília; Distrito Federal. |
Thesagro: |
Água; Alho; Allium Sativum; Cerrado; Deficiência; Irrigação; Solo. |
Thesaurus NAL: |
Brazil; garlic; irrigation; soil; water. |
Categoria do assunto: |
-- |
URL: |
https://www.scielo.br/scielo.php?script=sci_pdf&pid=S0102-05362002000300014&lng=pt&nrm=iso&tlng=pt
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Marc: |
LEADER 00840naa a2200313 a 4500 001 1772544 005 2002-11-13 008 2002 bl uuuu u00u1 u #d 100 1 $aMAROUELLI, W. A. 245 $aDesenvolvimento de plantas, produção e qualidade de bulbos de alho sob condições de deficiência de água no solo. 260 $c2002 650 $aBrazil 650 $agarlic 650 $airrigation 650 $asoil 650 $awater 650 $aÁgua 650 $aAlho 650 $aAllium Sativum 650 $aCerrado 650 $aDeficiência 650 $aIrrigação 650 $aSolo 653 $aBrasil 653 $aBrasília 653 $aDistrito Federal 700 1 $aSILVA, W. L. C. 700 1 $aMORETTI, C. L. 773 $tHorticultura Brasileira, Brasília, DF$gv. 20, n. 3, p. 470-473, set. 2002.
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