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Registro Completo |
Biblioteca(s): |
Embrapa Solos. |
Data corrente: |
10/03/2014 |
Data da última atualização: |
08/11/2021 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Autoria: |
FONSECA, E. M. da; BAPTISTA NETO, J. A.; MCALISTER, J.; SMITH, B.; FERNANDEZ, M. A.; BALIEIRO, F. C. |
Afiliação: |
ESTEFAN M. DA FONSECA, UFF; JOSÉ A. BAPTISTA NETO, UFF; JOHN MCALISTER, School of Geography; BERNARD SMITH, Queen’s University Belfast; MARCOS A. FERNANDEZ, UERJ; FABIANO DE CARVALHO BALIEIRO, CNPS. |
Título: |
The role of the humic substances in the fractioning of heavy metals in Rodrigo de Freitas Lagoon, Rio de Janeiro - Brazil. |
Ano de publicação: |
2014 |
Fonte/Imprenta: |
Anais da Academia Brasileira de Ciências, Rio de Janeiro, v. 85, n. 4, p. 1289-1301, 2014. |
DOI: |
https://doi.org/10.1590/0001-3765201371011 |
Idioma: |
Inglês |
Conteúdo: |
One of the main results of the processes related to urbanization is the contamination of the adjacent water bodies. Inserted in this context, the Rodrigo de Freitas lagoon is situated in the south zone of the city of Rio de Janeiro. This ecosystem receives several inputs containing all sorts of pollutants, including heavy metals. The present work aimed to study the partitioning of heavy metals in the sediments of Rodrigo de Freitas and the influence of organic matter in this fractionation dynamic. The results of these analyses presented the contents of organic matter as an important metal-capturing agent. Fractionation of organic matter resulted in a predominance of humine. Heavy metal partitioning showed that the metals bound by the water-soluble phase have no significant concentrations. Special features such as, reducing sediment, high levels of organic matter and fine grain size have transformed this ecosystem in an effective deposit of pollutants, where heavy metals are not available in easily reactive fractions. |
Palavras-Chave: |
Rio de Janeiro; Rodrigo de Freitas lagoon. |
Thesaurus Nal: |
Brazil; heavy metals; humic acids. |
Categoria do assunto: |
P Recursos Naturais, Ciências Ambientais e da Terra |
URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/98842/1/The-Role-of-the-Humic-Substances-in-the-Fractioning.pdf
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Marc: |
LEADER 01871naa a2200253 a 4500 001 1981933 005 2021-11-08 008 2014 bl uuuu u00u1 u #d 024 7 $ahttps://doi.org/10.1590/0001-3765201371011$2DOI 100 1 $aFONSECA, E. M. da 245 $aThe role of the humic substances in the fractioning of heavy metals in Rodrigo de Freitas Lagoon, Rio de Janeiro - Brazil.$h[electronic resource] 260 $c2014 520 $aOne of the main results of the processes related to urbanization is the contamination of the adjacent water bodies. Inserted in this context, the Rodrigo de Freitas lagoon is situated in the south zone of the city of Rio de Janeiro. This ecosystem receives several inputs containing all sorts of pollutants, including heavy metals. The present work aimed to study the partitioning of heavy metals in the sediments of Rodrigo de Freitas and the influence of organic matter in this fractionation dynamic. The results of these analyses presented the contents of organic matter as an important metal-capturing agent. Fractionation of organic matter resulted in a predominance of humine. Heavy metal partitioning showed that the metals bound by the water-soluble phase have no significant concentrations. Special features such as, reducing sediment, high levels of organic matter and fine grain size have transformed this ecosystem in an effective deposit of pollutants, where heavy metals are not available in easily reactive fractions. 650 $aBrazil 650 $aheavy metals 650 $ahumic acids 653 $aRio de Janeiro 653 $aRodrigo de Freitas lagoon 700 1 $aBAPTISTA NETO, J. A. 700 1 $aMCALISTER, J. 700 1 $aSMITH, B. 700 1 $aFERNANDEZ, M. A. 700 1 $aBALIEIRO, F. C. 773 $tAnais da Academia Brasileira de Ciências, Rio de Janeiro$gv. 85, n. 4, p. 1289-1301, 2014.
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Registro Completo
Biblioteca(s): |
Embrapa Meio Ambiente. |
Data corrente: |
15/01/2008 |
Data da última atualização: |
25/03/2014 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
Internacional - A |
Autoria: |
CASTRO, V. L. S. S. de; DESTEFANI, C. R.; DINIZ, C. A. R.; POLI, P. |
Afiliação: |
Vera Lúcia Scherholz Salgado de Castro, Embrapa Meio Ambiente; Camila R. Destefani, UFSCAR; Carlos Diniz, UFSCAR; Paola Poli, Università degli Studi di Parma. |
Título: |
Evaluation of neurodevelopmental effects on rats exposed prenatally to sulfentrazone. |
Ano de publicação: |
2007 |
Fonte/Imprenta: |
Neurotoxicology, Little Rock, v. 28, n. 6, p. 1249-1259, 2007. |
Idioma: |
Inglês |
Conteúdo: |
Although some studies have pointed to embryo/fetal toxicity at treatment levels that were not maternally toxic, knowledge about the potential toxic effects of the herbicide sulfentrazone is still limited. Since the results of these studies have raised some concern, the present work studied the effects of sulfentrazone maternal exposure on the physical and neurobehavioral endpoints in the development of rat pups. To accomplish that, the effects of the herbicide sulfentrazone (25 and 50mg/kg) were examined at two different developmental stages in rats: during the first 6 days of gestation, or in the organogenesis period (6-15 days). After parturition, pups were tested in a developmental test battery including measures of growth, maturational milestones, and neurobehavioral development. Maternal exposure to the herbicide resulted in significant alterations of the postnatal age at which the developmental milestones of ear and eye opening and testes descent were observed. There was a reduced weight gain rate in pups and their mothers when treated during the gestational period at the highest dose tested. Also, the functional state of the rat pup nervous system at different stages of postnatal development showed some neurodevelopmental delays in righting reflex, negative geotaxis, grip response, and motor coordination-locomotion and rearing (21-90 days of life) in the treated groups. Herbicide genotoxicity was investigated in fresh leukocytes both in mothers and pups using the comet assay: the data did not show any significant genotoxic effect induced by the herbicide. The findings of this study emphasize that sulfentrazone maternal exposure may lead to some neuromuscular and behavioral deficits in nursing pups. MenosAlthough some studies have pointed to embryo/fetal toxicity at treatment levels that were not maternally toxic, knowledge about the potential toxic effects of the herbicide sulfentrazone is still limited. Since the results of these studies have raised some concern, the present work studied the effects of sulfentrazone maternal exposure on the physical and neurobehavioral endpoints in the development of rat pups. To accomplish that, the effects of the herbicide sulfentrazone (25 and 50mg/kg) were examined at two different developmental stages in rats: during the first 6 days of gestation, or in the organogenesis period (6-15 days). After parturition, pups were tested in a developmental test battery including measures of growth, maturational milestones, and neurobehavioral development. Maternal exposure to the herbicide resulted in significant alterations of the postnatal age at which the developmental milestones of ear and eye opening and testes descent were observed. There was a reduced weight gain rate in pups and their mothers when treated during the gestational period at the highest dose tested. Also, the functional state of the rat pup nervous system at different stages of postnatal development showed some neurodevelopmental delays in righting reflex, negative geotaxis, grip response, and motor coordination-locomotion and rearing (21-90 days of life) in the treated groups. Herbicide genotoxicity was investigated in fresh leukocytes both in mothers and pups using the come... Mostrar Tudo |
Palavras-Chave: |
Coordenação motora; Sistema nervoso central; Teste. |
Thesagro: |
Cobaia; Herbicida; Rato; Toxicologia; Toxidez. |
Categoria do assunto: |
-- |
Marc: |
LEADER 02425naa a2200253 a 4500 001 1015559 005 2014-03-25 008 2007 bl uuuu u00u1 u #d 100 1 $aCASTRO, V. L. S. S. de 245 $aEvaluation of neurodevelopmental effects on rats exposed prenatally to sulfentrazone. 260 $c2007 520 $aAlthough some studies have pointed to embryo/fetal toxicity at treatment levels that were not maternally toxic, knowledge about the potential toxic effects of the herbicide sulfentrazone is still limited. Since the results of these studies have raised some concern, the present work studied the effects of sulfentrazone maternal exposure on the physical and neurobehavioral endpoints in the development of rat pups. To accomplish that, the effects of the herbicide sulfentrazone (25 and 50mg/kg) were examined at two different developmental stages in rats: during the first 6 days of gestation, or in the organogenesis period (6-15 days). After parturition, pups were tested in a developmental test battery including measures of growth, maturational milestones, and neurobehavioral development. Maternal exposure to the herbicide resulted in significant alterations of the postnatal age at which the developmental milestones of ear and eye opening and testes descent were observed. There was a reduced weight gain rate in pups and their mothers when treated during the gestational period at the highest dose tested. Also, the functional state of the rat pup nervous system at different stages of postnatal development showed some neurodevelopmental delays in righting reflex, negative geotaxis, grip response, and motor coordination-locomotion and rearing (21-90 days of life) in the treated groups. Herbicide genotoxicity was investigated in fresh leukocytes both in mothers and pups using the comet assay: the data did not show any significant genotoxic effect induced by the herbicide. The findings of this study emphasize that sulfentrazone maternal exposure may lead to some neuromuscular and behavioral deficits in nursing pups. 650 $aCobaia 650 $aHerbicida 650 $aRato 650 $aToxicologia 650 $aToxidez 653 $aCoordenação motora 653 $aSistema nervoso central 653 $aTeste 700 1 $aDESTEFANI, C. R. 700 1 $aDINIZ, C. A. R. 700 1 $aPOLI, P. 773 $tNeurotoxicology, Little Rock$gv. 28, n. 6, p. 1249-1259, 2007.
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