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Registro Completo |
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Biblioteca(s): |
Embrapa Florestas. |
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Data corrente: |
21/10/2010 |
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Data da última atualização: |
01/02/2018 |
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Tipo da produção científica: |
Artigo em Periódico Indexado |
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Autoria: |
ROSADO, C. C. G.; GUIMARÃES, L. M. da S.; TITON, M.; LAU, D.; ROSSE, L.; RESENDE, M. D. V. de; ALFENAS, A. C. |
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Afiliação: |
CARLA CRISTINA GONÇALVES ROSADO, UFV; LÚCIO MAURO DA SILVA GUIMARÃES, UFV; MIRANDA TITON, UFV; DOUGLAS LAU, CNPT; LEONARDO ROSSE, VERACEL; MARCOS DEON VILELA DE RESENDE, CNPF; ACELINO COUTO ALFENAS, UFV. |
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Título: |
Resistance to ceratocystis wilt (Ceratocystis fimbriata) in parents and progenies of Eucalyptus grandis x E. urophylla. |
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Ano de publicação: |
2010 |
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Fonte/Imprenta: |
Silvae Genetica, v. 59, n. 2/3, p. 99-106, 2010. |
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Idioma: |
Inglês |
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Conteúdo: |
Ceratocystis wilt, caused by Ceratocystis fimbriata, is one ofthe most damaging diseases in eucalyptus plantations worldwide. Although there are resistant genotypes, the genetic basis of resistance is still poorly understood. In this paper we studied the resistance leveI by a stem inoculation experiment of genotypes of Eucalyptus grandis and E. urophylla and estimated the heritability and gains of selection in families derived from controlled interspecific crosses. In both species, highly resistant as well as highly susceptible genotypes to Ceratocystis wilt were found. Out of 21 parents assessed, twelve were resistant and nine susceptible. Estimates of individual narrow (50%) and broad (59%) sense heritability suggested a high degree of genetic control and low allelic dominance of the trait. There was great genetic variation among and within families, a fact that contributes to high heritability and genetic gain. A genetic gain in lesion size of up to -74.4 % was obtained from selection of the 50 best clones in the evaluated families, i.e., the mean lesion length in the progeny population can be reduced by 74,4%. |
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Thesagro: |
Ceratocystis Fimbriata; Doença; Eucalipto; Resistência. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/34116/1/RESISTANCES-TO-CERATOCYSTIS-WILT.pdf
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Marc: |
LEADER 01827naa a2200241 a 4500
001 1864849
005 2018-02-01
008 2010 bl uuuu u00u1 u #d
100 1 $aROSADO, C. C. G.
245 $aResistance to ceratocystis wilt (Ceratocystis fimbriata) in parents and progenies of Eucalyptus grandis x E. urophylla.
260 $c2010
520 $aCeratocystis wilt, caused by Ceratocystis fimbriata, is one ofthe most damaging diseases in eucalyptus plantations worldwide. Although there are resistant genotypes, the genetic basis of resistance is still poorly understood. In this paper we studied the resistance leveI by a stem inoculation experiment of genotypes of Eucalyptus grandis and E. urophylla and estimated the heritability and gains of selection in families derived from controlled interspecific crosses. In both species, highly resistant as well as highly susceptible genotypes to Ceratocystis wilt were found. Out of 21 parents assessed, twelve were resistant and nine susceptible. Estimates of individual narrow (50%) and broad (59%) sense heritability suggested a high degree of genetic control and low allelic dominance of the trait. There was great genetic variation among and within families, a fact that contributes to high heritability and genetic gain. A genetic gain in lesion size of up to -74.4 % was obtained from selection of the 50 best clones in the evaluated families, i.e., the mean lesion length in the progeny population can be reduced by 74,4%.
650 $aCeratocystis Fimbriata
650 $aDoença
650 $aEucalipto
650 $aResistência
700 1 $aGUIMARÃES, L. M. da S.
700 1 $aTITON, M.
700 1 $aLAU, D.
700 1 $aROSSE, L.
700 1 $aRESENDE, M. D. V. de
700 1 $aALFENAS, A. C.
773 $tSilvae Genetica$gv. 59, n. 2/3, p. 99-106, 2010.
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Registro original: |
Embrapa Florestas (CNPF) |
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Registro Completo
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Biblioteca(s): |
Embrapa Unidades Centrais. |
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Data corrente: |
02/02/2018 |
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Data da última atualização: |
01/08/2018 |
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Autoria: |
SILVA, V. C.; OLIVEIRA, L. A. de; LACERDA, M. dos S. C.; PIMENTEL, L. A.; SANTOS, W. S.; MACÊDO, J. T. S. A. e; RIET-CORREA, F.; PEDROSO P. M. O. |
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Afiliação: |
Valdir C. Silva, Setor de Patologia Veterinária/Hospital Universitário de Medicina Veterinária/Universidade Federal do Recôncavo da Bahia - UFRB; LUCIANA ALVES DE OLIVEIRA, CNPMF; Maira dos S.C. Lacerda, Setor de Patologia Veterinária/Hospital Universitário de Medicina Veterinária/Universidade Federal do Recôncavo da Bahia - UFRB; Luciano A. Pimentel, Setor de Patologia Veterinária/Hospital Universitário de Medicina Veterinária/Universidade Federal do Recôncavo da Bahia - UFRB; Wiles S. Santos, Setor de Patologia Veterinária/Hospital Universitário de Medicina Veterinária/Universidade Federal do Recôncavo da Bahia - UFRB; Juliana Targino S.A. e Macêdo, Laboratório de Patologia Veterinária/Fundação Universidade de Brasília - UnB; Franklin Riet-Correa; Pedro M.O. Pedroso, Laboratório de Patologia Veterinária/Fundação Universidade de Brasília - UnB. |
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Título: |
Experimental poisoning by cassava wastewater in sheep. |
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Ano de publicação: |
2017 |
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Fonte/Imprenta: |
Pesquisa Veterinária Brasileira, Rio de Janeiro, v. 37, n. 11, p. 1241-1246, novembro. 2017. |
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Idioma: |
Inglês |
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Notas: |
Título em português: Intoxicação experimental por manipueira em ovinos. |
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Conteúdo: |
The processing of Manihot esculenta (cassava) tubers yield different by-products, including cassava wastewater, which is the liquid pressed out of the tuber after it has been mechanically crushed. Cyanide poisoning after ingestion of cassava wastewater has been reported in ruminants and pigs in Northeastern Brazil. With the aim of studying its toxicity, cassava wastewater was administered orally to six sheep at doses of 0.99, 0.75, 0.70, 0.63, and 0.5 mg of hydrocyanic acid kg-1 body weight, which corresponded to 14.2, 10.6, 9.8, 8.89, and 7.1 mL of wastewater kg-1. On the second day, the sheep received a volume of wastewater which corresponded to 0.46, 0.34, 0.31, 0.28, and 0.23 mg of HCN kg-1. A sheep used as control received 9.9 mL of water kg-1 BW. Sheep that received from 0.75 to 0.99 mg kg-1 of HCN on the first day exhibited severe clinical signs of poisoning, and the sheep that received 0.63 and 0.5 mg kg-1 exhibited mild clinical signs. All sheep were successfully treated with sodium thiosulfate. On the second day, only the sheep that received 0.46 mg kg-1 and 0.34 mg kg-1 exhibited mild clinical signs and recovered spontaneously. The concentration of HCN in the wastewater was 71.69±2.19 ?g mL-1 immediately after production, 30.56±2.45 ?g mL-1 after 24 hours, and 24.25±1.28 ?g mL-1 after 48 hours. The picric acid paper test was strongly positive 5 minutes after production; moderately positive 24 hours after production, and negative 48 hours after production. We conclude that cassava wastewater is highly toxic to sheep if ingested immediately after production, but rapidly loses toxicity in 24-48 hours. MenosThe processing of Manihot esculenta (cassava) tubers yield different by-products, including cassava wastewater, which is the liquid pressed out of the tuber after it has been mechanically crushed. Cyanide poisoning after ingestion of cassava wastewater has been reported in ruminants and pigs in Northeastern Brazil. With the aim of studying its toxicity, cassava wastewater was administered orally to six sheep at doses of 0.99, 0.75, 0.70, 0.63, and 0.5 mg of hydrocyanic acid kg-1 body weight, which corresponded to 14.2, 10.6, 9.8, 8.89, and 7.1 mL of wastewater kg-1. On the second day, the sheep received a volume of wastewater which corresponded to 0.46, 0.34, 0.31, 0.28, and 0.23 mg of HCN kg-1. A sheep used as control received 9.9 mL of water kg-1 BW. Sheep that received from 0.75 to 0.99 mg kg-1 of HCN on the first day exhibited severe clinical signs of poisoning, and the sheep that received 0.63 and 0.5 mg kg-1 exhibited mild clinical signs. All sheep were successfully treated with sodium thiosulfate. On the second day, only the sheep that received 0.46 mg kg-1 and 0.34 mg kg-1 exhibited mild clinical signs and recovered spontaneously. The concentration of HCN in the wastewater was 71.69±2.19 ?g mL-1 immediately after production, 30.56±2.45 ?g mL-1 after 24 hours, and 24.25±1.28 ?g mL-1 after 48 hours. The picric acid paper test was strongly positive 5 minutes after production; moderately positive 24 hours after production, and negative 48 hours after production. We concl... Mostrar Tudo |
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Palavras-Chave: |
Ácido cianídrico; Nordeste do Brasil; Northeastern Brazil. |
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Thesagro: |
Acido prussico; Mandioca; Manipueira; Ovino; Ruminante. |
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Thesaurus NAL: |
Cassava; Hydrogen cyanide; Ruminants; Sheep; Wastewater. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/172111/1/Experimental-poisoning-by-cassava-wastewater-in-sheep.pdf
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Marc: |
LEADER 02733naa a2200373 a 4500
001 2087001
005 2018-08-01
008 2017 bl uuuu u00u1 u #d
100 1 $aSILVA, V. C.
245 $aExperimental poisoning by cassava wastewater in sheep.$h[electronic resource]
260 $c2017
500 $aTítulo em português: Intoxicação experimental por manipueira em ovinos.
520 $aThe processing of Manihot esculenta (cassava) tubers yield different by-products, including cassava wastewater, which is the liquid pressed out of the tuber after it has been mechanically crushed. Cyanide poisoning after ingestion of cassava wastewater has been reported in ruminants and pigs in Northeastern Brazil. With the aim of studying its toxicity, cassava wastewater was administered orally to six sheep at doses of 0.99, 0.75, 0.70, 0.63, and 0.5 mg of hydrocyanic acid kg-1 body weight, which corresponded to 14.2, 10.6, 9.8, 8.89, and 7.1 mL of wastewater kg-1. On the second day, the sheep received a volume of wastewater which corresponded to 0.46, 0.34, 0.31, 0.28, and 0.23 mg of HCN kg-1. A sheep used as control received 9.9 mL of water kg-1 BW. Sheep that received from 0.75 to 0.99 mg kg-1 of HCN on the first day exhibited severe clinical signs of poisoning, and the sheep that received 0.63 and 0.5 mg kg-1 exhibited mild clinical signs. All sheep were successfully treated with sodium thiosulfate. On the second day, only the sheep that received 0.46 mg kg-1 and 0.34 mg kg-1 exhibited mild clinical signs and recovered spontaneously. The concentration of HCN in the wastewater was 71.69±2.19 ?g mL-1 immediately after production, 30.56±2.45 ?g mL-1 after 24 hours, and 24.25±1.28 ?g mL-1 after 48 hours. The picric acid paper test was strongly positive 5 minutes after production; moderately positive 24 hours after production, and negative 48 hours after production. We conclude that cassava wastewater is highly toxic to sheep if ingested immediately after production, but rapidly loses toxicity in 24-48 hours.
650 $aCassava
650 $aHydrogen cyanide
650 $aRuminants
650 $aSheep
650 $aWastewater
650 $aAcido prussico
650 $aMandioca
650 $aManipueira
650 $aOvino
650 $aRuminante
653 $aÁcido cianídrico
653 $aNordeste do Brasil
653 $aNortheastern Brazil
700 1 $aOLIVEIRA, L. A. de
700 1 $aLACERDA, M. dos S. C.
700 1 $aPIMENTEL, L. A.
700 1 $aSANTOS, W. S.
700 1 $aMACÊDO, J. T. S. A. e
700 1 $aRIET-CORREA, F.
700 1 $aPEDROSO P. M. O.
773 $tPesquisa Veterinária Brasileira, Rio de Janeiro$gv. 37, n. 11, p. 1241-1246, novembro. 2017.
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Embrapa Unidades Centrais (AI-SEDE) |
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