02048naa a2200205 a 450000100080000000500110000800800410001910000230006024500590008326000090014230000110015149000370016250001800019952011440037970000200152370000200154370000180156370000150158177302460159614667682007-07-27       2004    bl uuuu     u00u1 u    #d1 aKLINGELFUSS, L. H.  aSoybean heritance to sudden death syndrome resistance.  c2004  ap. 98.  a(Embrapa Soja. Documentos, 228).  aEditado por Flávio Moscardi, Clara Beatriz Hoffmann-Campo, Odilon Ferreira Saraiva, Paulo Roberto Galerani, Francisco Carlos Krzyzanowski, Mercedes Concordia Carrão-Panizzi.  aOver the past few years yield losses caused by soybean diseases have become more and more serious, demanding the development of new technologies of control. Sudden death syndrome (SDS) is a fungal disease caused by Fusarium solani f. sp. glycines, that has been considered of great relevance due to the substantial soybean yield reductions and control limitations. Control has been possible by means of resistant genes. This work aims to study screening for resistance to SDS. After being inoculated with the pathogen, soybean plants originated from the crosses of FT Estrela, Ripley, MG/BR 46 - Conquista and PI 520733 besides their parental generations, F1, F2 and F3, including the reciprocals, were assessed in the experiment. Genetic models were adjusted to the means and variances of the generations. Models including significant genetic effects were set only in the crosses containing PI 520733, where the additive effect was predominant. The heredity ranged from average to low. The screening for resistance must be always conducted under controlled conditions and based on progenies, resulting in gains from the early generations.1 aYORINORI, J. T.1 aARIAS, C. A. A.1 aBROGIN, R. L.1 aDESTRO, D.  tIn: WORLD SOYBEAN RESEARCH CONFERENCE, 7.; INTERNATIONAL SOYBEAN PROCESSING AND UTILIZATION CONFERENCE, 4.; CONGRESSO BRASILEIRO DE SOJA, 3., 2004, Foz do Iguassu. Abstracts of contributed papers and posters. Londrina: Embrapa Soybean, 2004.