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Registro Completo |
Biblioteca(s): |
Embrapa Milho e Sorgo. |
Data corrente: |
23/01/2006 |
Data da última atualização: |
29/05/2018 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Autoria: |
NINAMANGO-CARDENAS, F. E.; GUIMARAES, C. T.; MARTINS, P. R.; PARENTONI, S. N.; CARNEIRO, N. P.; LOPES, M. A.; MORO, J. R.; PAIVA, E. |
Afiliação: |
CLAUDIA TEIXEIRA GUIMARAES, CNPMS; SIDNEY NETTO PARENTONI, CNPMS; NEWTON PORTILHO CARNEIRO, CNPMS. |
Título: |
Mapping QTLs for aluminum tolerance in maize. |
Ano de publicação: |
2003 |
Fonte/Imprenta: |
Euphytica, Wageningen, v. 130, n. 2 p. 223-232, 2003. |
Idioma: |
Inglês |
Conteúdo: |
Aluminum toxicity is one of the major constraints for plant development in acid soils, limiting food production in many countries. Cultivars genetically adapted to acid soils may offer an environmental compatible solution, providing a sustainable agriculture system. The aim of this work was to identify genomic regions associated with Al tolerance in maize, and to quantify the genetic effects on the phenotypic variation. A population of 168 F3:4 families derived from a cross between two contrasting maize inbred lines for Al tolerance was evaluated using the NSRL and RSRL parameters in nutrient solution containing toxic level of aluminum. Variance analyses indicated that the NSRL was the most reliable phenotypic index to measure Al tolerance in the population, being used for further QTL mapping analysis. RFLP and SSR markers were selected for bulked segregant analysis, and additional SSR markers, flanking the polymorphisms of interest, were chosen in order to saturate the putative target regions. Seven linkage groups were constructed using 17 RFLP and 34 SSR markers. Five QTLs were mapped on chromosomes 2, 6 and 8, explaining 60% of the phenotypic variation. QTL4 and marker umc043 were located on chromosomes 8 and 5, close to genes encoding for enzymes involved in the organic acids synthesis pathways, a widely proposed mechanism for Al tolerance in plants. QTL2 was mapped in the same region as Alm2, also associated with Al tolerance in maize. In addition, dominant and additive effects were important in the control of this trait in maize.. MenosAluminum toxicity is one of the major constraints for plant development in acid soils, limiting food production in many countries. Cultivars genetically adapted to acid soils may offer an environmental compatible solution, providing a sustainable agriculture system. The aim of this work was to identify genomic regions associated with Al tolerance in maize, and to quantify the genetic effects on the phenotypic variation. A population of 168 F3:4 families derived from a cross between two contrasting maize inbred lines for Al tolerance was evaluated using the NSRL and RSRL parameters in nutrient solution containing toxic level of aluminum. Variance analyses indicated that the NSRL was the most reliable phenotypic index to measure Al tolerance in the population, being used for further QTL mapping analysis. RFLP and SSR markers were selected for bulked segregant analysis, and additional SSR markers, flanking the polymorphisms of interest, were chosen in order to saturate the putative target regions. Seven linkage groups were constructed using 17 RFLP and 34 SSR markers. Five QTLs were mapped on chromosomes 2, 6 and 8, explaining 60% of the phenotypic variation. QTL4 and marker umc043 were located on chromosomes 8 and 5, close to genes encoding for enzymes involved in the organic acids synthesis pathways, a widely proposed mechanism for Al tolerance in plants. QTL2 was mapped in the same region as Alm2, also associated with Al tolerance in maize. In addition, dominant and additive... Mostrar Tudo |
Palavras-Chave: |
chromosomes-; Cromossomo; Mapeamento genético. |
Thesagro: |
Alumínio; Genética; Marcador Genético; Milho. |
Categoria do assunto: |
-- |
Marc: |
LEADER 02333naa a2200289 a 4500 001 1489128 005 2018-05-29 008 2003 bl uuuu u00u1 u #d 100 1 $aNINAMANGO-CARDENAS, F. E. 245 $aMapping QTLs for aluminum tolerance in maize.$h[electronic resource] 260 $c2003 520 $aAluminum toxicity is one of the major constraints for plant development in acid soils, limiting food production in many countries. Cultivars genetically adapted to acid soils may offer an environmental compatible solution, providing a sustainable agriculture system. The aim of this work was to identify genomic regions associated with Al tolerance in maize, and to quantify the genetic effects on the phenotypic variation. A population of 168 F3:4 families derived from a cross between two contrasting maize inbred lines for Al tolerance was evaluated using the NSRL and RSRL parameters in nutrient solution containing toxic level of aluminum. Variance analyses indicated that the NSRL was the most reliable phenotypic index to measure Al tolerance in the population, being used for further QTL mapping analysis. RFLP and SSR markers were selected for bulked segregant analysis, and additional SSR markers, flanking the polymorphisms of interest, were chosen in order to saturate the putative target regions. Seven linkage groups were constructed using 17 RFLP and 34 SSR markers. Five QTLs were mapped on chromosomes 2, 6 and 8, explaining 60% of the phenotypic variation. QTL4 and marker umc043 were located on chromosomes 8 and 5, close to genes encoding for enzymes involved in the organic acids synthesis pathways, a widely proposed mechanism for Al tolerance in plants. QTL2 was mapped in the same region as Alm2, also associated with Al tolerance in maize. In addition, dominant and additive effects were important in the control of this trait in maize.. 650 $aAlumínio 650 $aGenética 650 $aMarcador Genético 650 $aMilho 653 $achromosomes- 653 $aCromossomo 653 $aMapeamento genético 700 1 $aGUIMARAES, C. T. 700 1 $aMARTINS, P. R. 700 1 $aPARENTONI, S. N. 700 1 $aCARNEIRO, N. P. 700 1 $aLOPES, M. A. 700 1 $aMORO, J. R. 700 1 $aPAIVA, E. 773 $tEuphytica, Wageningen$gv. 130, n. 2 p. 223-232, 2003.
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Embrapa Milho e Sorgo (CNPMS) |
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Biblioteca(s): |
Embrapa Agricultura Digital. |
Data corrente: |
29/05/2012 |
Data da última atualização: |
22/05/2024 |
Tipo da produção científica: |
Artigo em Periódico Indexado |
Circulação/Nível: |
A - 1 |
Autoria: |
FARIA, L. C. B.; ROCHA, A. S. L.; KLEINSCHMIDT, J. H.; SILVA-FILHO, M. C.; BIM, E.; HERAI, R. H.; YAMAGISHI, M. E. B.; PALAZZO JÚNIOR, R. |
Afiliação: |
LUZINETE C. B. FARIA, UNIVERSIDADE ESTADUAL DE CAMPINAS; ANDRÉA S. L. ROCHA, UNIVERSIDADE ESTADUAL DE CAMPINAS; JOÃO H. KLEINSCHMIDT, UNIVERSIDADE FEDERAL DO ABC; MÁRCIO C. SILVA-FILHO, UNIVERSIDADE DE SÃO PAULO; EDSON BIM, UNIVERSIDADE ESTADUAL DE CAMPINAS; ROBERTO H. HERAI, UNIVERSITY OF CALIFORNIA; MICHEL EDUARDO BELEZA YAMAGISHI, CNPTIA; REGINALDO PALAZZO JÚNIOR, UNIVERSIDADE ESTADUAL DE CAMPINAS. |
Título: |
Is a genome a codeword of an error-correcting code? |
Ano de publicação: |
2012 |
Fonte/Imprenta: |
PLoS ONE, San Francisco, v. 7, n. 5, e105396, May 2012. |
DOI: |
https://doi.org/10.1371/journal.pone.0036644 |
Idioma: |
Inglês |
Conteúdo: |
Since a genome is a discrete sequence, the elements of which belong to a set of four letters, the question as to whether or not there is an error-correcting code underlying DNA sequences is unavoidable. The most common approach to answering this question is to propose a methodology to verify the existence of such a code. However, none of the methodologies proposed so far, although quite clever, has achieved that goal. In a recent work, we showed that DNA sequences can be identified as codewords in a class of cyclic error-correcting codes known as Hamming codes. In this paper, we show that a complete intron-exon gene, and even a plasmid genome, can be identified as a Hamming code codeword as well. Although this does not constitute a definitive proof that there is an error-correcting code underlying DNA sequences, it is the first evidence in this direction. |
Palavras-Chave: |
Biology; Sequência de DNA. |
Thesagro: |
Biologia. |
Thesaurus NAL: |
Genome; Nucleotide sequences. |
Categoria do assunto: |
-- |
URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/60323/1/journal.pone.0036644.pdf
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Marc: |
LEADER 01659naa a2200277 a 4500 001 1925637 005 2024-05-22 008 2012 bl uuuu u00u1 u #d 024 7 $ahttps://doi.org/10.1371/journal.pone.0036644$2DOI 100 1 $aFARIA, L. C. B. 245 $aIs a genome a codeword of an error-correcting code?$h[electronic resource] 260 $c2012 520 $aSince a genome is a discrete sequence, the elements of which belong to a set of four letters, the question as to whether or not there is an error-correcting code underlying DNA sequences is unavoidable. The most common approach to answering this question is to propose a methodology to verify the existence of such a code. However, none of the methodologies proposed so far, although quite clever, has achieved that goal. In a recent work, we showed that DNA sequences can be identified as codewords in a class of cyclic error-correcting codes known as Hamming codes. In this paper, we show that a complete intron-exon gene, and even a plasmid genome, can be identified as a Hamming code codeword as well. Although this does not constitute a definitive proof that there is an error-correcting code underlying DNA sequences, it is the first evidence in this direction. 650 $aGenome 650 $aNucleotide sequences 650 $aBiologia 653 $aBiology 653 $aSequência de DNA 700 1 $aROCHA, A. S. L. 700 1 $aKLEINSCHMIDT, J. H. 700 1 $aSILVA-FILHO, M. C. 700 1 $aBIM, E. 700 1 $aHERAI, R. H. 700 1 $aYAMAGISHI, M. E. B. 700 1 $aPALAZZO JÚNIOR, R. 773 $tPLoS ONE, San Francisco$gv. 7, n. 5, e105396, May 2012.
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