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Registro Completo
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Biblioteca(s): |
Embrapa Pantanal. |
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Data corrente: |
11/09/1997 |
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Data da última atualização: |
11/09/1997 |
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Autoria: |
SILVA, R. A. M. S.; DAVILA, A. M. R.; AROSEMENA, N. A. E. |
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Afiliação: |
EMBRAPA. Centro de Pesquisa Agropecuaria do Pantanal (Corumba, MS). |
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Título: |
Outbreak of equine Trypanosomosis due to Trypanosoma evansi in the Pantanal, Brazil. |
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Ano de publicação: |
1995 |
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Fonte/Imprenta: |
Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.90, p.297, 1995. Suplemento 1. |
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Idioma: |
Inglês |
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Conteúdo: |
The Pantanal is a wetland with an area of 140.000 km2, located in the centre of South America. The Pantanal can be considered one of the moswt important livestock production centers in Brazil. T. evansi probably was introduced into the region the 16 th century by Spanish settlers. However we are reporting here for the first time trypanosomosis due to T. evansi in horses of Pantanal. In February 1994 an outbreak of equine trypanosomosis occurred in a ranch with forty-eight dead horses (51%). The clinical signs observed were fever, anemia, conjunctivitis, edema of the legs and lower parts of the body, progressive weakness a gradual onset of paresis of hind quarters, loss of condition and inappetence. The diagnosis was confirmed by morphological and biometrical studies. The animals were bled for determination of hematological data using a vacuum system in tubes EDTA as anti-coagulant. The T. evansi morphological and biometrical data were obtained from blood sears of naturally infected horses. The blood smears were examined after staining with May-Gruenwald-Giemsa. The isolates were identified based on morphological and biometrical data. The blood values observed were: 3.65 x 10 6 RBC, 6.92 x 10 3 WBC, 18% PCV, 5.53g/dl Hb, 15.15 pg mean corpuscular hemoglobin and 30.72% mean corpuscular hemoglobin concentration. The differential white cell counts were: 6.6% neutrophil, 60.2% lymphocyte, 29% monocyte, 4.2% eosinophi. In our study the infected animalsshoweda marked decrease in RBC, Hb and PCV. The increase in absolute lymphocyte count observed in sick horses has been reported in trypanosomosis by T. evansi and infections caused by other trypanosomes. Measurements of T. evansi of equines from Pantanal, Brazil, means +- SE (mm)(n=100). MenosThe Pantanal is a wetland with an area of 140.000 km2, located in the centre of South America. The Pantanal can be considered one of the moswt important livestock production centers in Brazil. T. evansi probably was introduced into the region the 16 th century by Spanish settlers. However we are reporting here for the first time trypanosomosis due to T. evansi in horses of Pantanal. In February 1994 an outbreak of equine trypanosomosis occurred in a ranch with forty-eight dead horses (51%). The clinical signs observed were fever, anemia, conjunctivitis, edema of the legs and lower parts of the body, progressive weakness a gradual onset of paresis of hind quarters, loss of condition and inappetence. The diagnosis was confirmed by morphological and biometrical studies. The animals were bled for determination of hematological data using a vacuum system in tubes EDTA as anti-coagulant. The T. evansi morphological and biometrical data were obtained from blood sears of naturally infected horses. The blood smears were examined after staining with May-Gruenwald-Giemsa. The isolates were identified based on morphological and biometrical data. The blood values observed were: 3.65 x 10 6 RBC, 6.92 x 10 3 WBC, 18% PCV, 5.53g/dl Hb, 15.15 pg mean corpuscular hemoglobin and 30.72% mean corpuscular hemoglobin concentration. The differential white cell counts were: 6.6% neutrophil, 60.2% lymphocyte, 29% monocyte, 4.2% eosinophi. In our study the infected animalsshoweda marked decrease in RB... Mostrar Tudo |
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Palavras-Chave: |
Equine. |
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Thesagro: |
Eqüino. |
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Thesaurus NAL: |
Brazil; Pantanal; Trypanosoma evansi. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 02354naa a2200205 a 4500
001 1791635
005 1997-09-11
008 1995 bl uuuu u00u1 u #d
100 1 $aSILVA, R. A. M. S.
245 $aOutbreak of equine Trypanosomosis due to Trypanosoma evansi in the Pantanal, Brazil.
260 $c1995
520 $aThe Pantanal is a wetland with an area of 140.000 km2, located in the centre of South America. The Pantanal can be considered one of the moswt important livestock production centers in Brazil. T. evansi probably was introduced into the region the 16 th century by Spanish settlers. However we are reporting here for the first time trypanosomosis due to T. evansi in horses of Pantanal. In February 1994 an outbreak of equine trypanosomosis occurred in a ranch with forty-eight dead horses (51%). The clinical signs observed were fever, anemia, conjunctivitis, edema of the legs and lower parts of the body, progressive weakness a gradual onset of paresis of hind quarters, loss of condition and inappetence. The diagnosis was confirmed by morphological and biometrical studies. The animals were bled for determination of hematological data using a vacuum system in tubes EDTA as anti-coagulant. The T. evansi morphological and biometrical data were obtained from blood sears of naturally infected horses. The blood smears were examined after staining with May-Gruenwald-Giemsa. The isolates were identified based on morphological and biometrical data. The blood values observed were: 3.65 x 10 6 RBC, 6.92 x 10 3 WBC, 18% PCV, 5.53g/dl Hb, 15.15 pg mean corpuscular hemoglobin and 30.72% mean corpuscular hemoglobin concentration. The differential white cell counts were: 6.6% neutrophil, 60.2% lymphocyte, 29% monocyte, 4.2% eosinophi. In our study the infected animalsshoweda marked decrease in RBC, Hb and PCV. The increase in absolute lymphocyte count observed in sick horses has been reported in trypanosomosis by T. evansi and infections caused by other trypanosomes. Measurements of T. evansi of equines from Pantanal, Brazil, means +- SE (mm)(n=100).
650 $aBrazil
650 $aPantanal
650 $aTrypanosoma evansi
650 $aEqüino
653 $aEquine
700 1 $aDAVILA, A. M. R.
700 1 $aAROSEMENA, N. A. E.
773 $tMemorias do Instituto Oswaldo Cruz, Rio de Janeiro$gv.90, p.297, 1995. Suplemento 1.
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