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Registro Completo |
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Biblioteca(s): |
Embrapa Clima Temperado. |
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Data corrente: |
05/12/2012 |
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Data da última atualização: |
18/02/2016 |
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Tipo da produção científica: |
Resumo em Anais de Congresso |
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Autoria: |
MISTURA, C. C.; BARBIERI, R. L.; CASTRO, C. M.; CARBONARI, T.; FONSECA, M. de. |
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Afiliação: |
Claudete Clarice Mistura, Eng" Agrônoma, M.Sc. em Agronomia. Universidade Federal de Pelotas.; ROSA LIA BARBIERI, CPACT; CAROLINE MARQUES CASTRO, CPACT; Taíse Carbonari, Estudante de Agronomia, Universidade Federal de Pelotas.; Marina da Fonsecas, Bióloga. estudante de Mestrado em Agronomia, Universidade Federal de Pelotas. |
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Título: |
Caracterização de uma população natural de butia odorata (barb. rodr.) noblick & lorenzi: cor e tamanho de frutos. |
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Ano de publicação: |
2012 |
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Fonte/Imprenta: |
In:SIMPÓSIO NACIONAL DO MORANGO, 6.; ENCONTRO SOBRE PEQUENAS FRUTAS E FRUTAS NATIVAS DO MERCOSUL, 5., 2012, Pelotas. Palestras e resumos... Pelotas: Embrapa Clima Temperado, 2012. 231 p. Organizadores: Marcia Vizzotto, Carlos Reisser Júnior, Rosa Lia Barbieri, Rodrigo Cezar Franzon. |
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Idioma: |
Português |
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Palavras-Chave: |
NATURAL. |
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Categoria do assunto: |
X Pesquisa, Tecnologia e Engenharia |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/item/71545/1/Digitalizar0022.pdf
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Marc: |
LEADER 00766nam a2200157 a 4500
001 1941419
005 2016-02-18
008 2012 bl uuuu u00u1 u #d
100 1 $aMISTURA, C. C.
245 $aCaracterização de uma população natural de butia odorata (barb. rodr.) noblick & lorenzi$bcor e tamanho de frutos.$h[electronic resource]
260 $aIn:SIMPÓSIO NACIONAL DO MORANGO, 6.; ENCONTRO SOBRE PEQUENAS FRUTAS E FRUTAS NATIVAS DO MERCOSUL, 5., 2012, Pelotas. Palestras e resumos... Pelotas: Embrapa Clima Temperado, 2012. 231 p. Organizadores: Marcia Vizzotto, Carlos Reisser Júnior, Rosa Lia Barbieri, Rodrigo Cezar Franzon.$c2012
653 $aNATURAL
700 1 $aBARBIERI, R. L.
700 1 $aCASTRO, C. M.
700 1 $aCARBONARI, T.
700 1 $aFONSECA, M. de
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Embrapa Clima Temperado (CPACT) |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Pantanal. Para informações adicionais entre em contato com cpap.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Pantanal. |
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Data corrente: |
19/03/1997 |
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Data da última atualização: |
31/03/2017 |
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Autoria: |
RIVERA DAVILA, A. M.; SILVA, R. A. M. S.; RAMIREZ, L. |
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Afiliação: |
UFMS. Centro Universitario de Corumba (Corumba, MS); EMBRAPA. Centro de Pesquisa Agropecuaria do Pantanal (Corumba, MS). |
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Título: |
Protection from lethal infection by prior immunization with an homologous Trypanosoma evansi Strain. |
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Ano de publicação: |
1995 |
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Fonte/Imprenta: |
In: CONGRESSO DA SOCIEDADE BRASILEIRA DE PARASITOLOGIA, 14., 1995, Goiania. Resumos... Goiania: [s.n.] 1995. p.325. |
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Idioma: |
Inglês |
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Conteúdo: |
Trypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance of trypomastigotes was successful on 1 day-post-quimiotherapy (d.p.q.) Seventeen d.p.q. rats were re-chanllenge with log. 7.69 trypomastigotes of homologous variant (double dose of first challenge), protection was successful during 49 after first-challenge, when animals were re-challenge with log 8.47 trypamastigotes of homologous variant (1345% more first inocolum) obtained of sucessive syringe passaged in others Wistar rats. Partial protection was observed, prepatent period was of 4 days, and log. 67,7/ml (in 75% of rats) and log. 8.52/ml (in 25% of rats) on the first and 9th day of parasitemia were respectivily registred. Control group parasitemia was log 6.38/ml and log 8.7/ml on first and 5th d.p.i. (died). Possible antigenic variation of strain on the third challenge or overdose of inocolum are postuled. Serum sample were collected during experiment for after evaluation. We verificated protection conferred against homologous T. evansi strain, as demonstred by others authors with T. congolense, T. brucei, T. cruzi and T. evansi. More studies are necessary to determinate which mechanisms are involved in the partial protection. MenosTrypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance ... Mostrar Tudo |
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Palavras-Chave: |
Infeccao letal; Lethal infection. |
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Thesagro: |
Imunização. |
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Thesaurus NAL: |
immunization; Pantanal; Trypanosoma evansi. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 03338naa a2200217 a 4500
001 1790947
005 2017-03-31
008 1995 bl --- 0-- u #d
100 1 $aRIVERA DAVILA, A. M.
245 $aProtection from lethal infection by prior immunization with an homologous Trypanosoma evansi Strain.
260 $c1995
520 $aTrypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance of trypomastigotes was successful on 1 day-post-quimiotherapy (d.p.q.) Seventeen d.p.q. rats were re-chanllenge with log. 7.69 trypomastigotes of homologous variant (double dose of first challenge), protection was successful during 49 after first-challenge, when animals were re-challenge with log 8.47 trypamastigotes of homologous variant (1345% more first inocolum) obtained of sucessive syringe passaged in others Wistar rats. Partial protection was observed, prepatent period was of 4 days, and log. 67,7/ml (in 75% of rats) and log. 8.52/ml (in 25% of rats) on the first and 9th day of parasitemia were respectivily registred. Control group parasitemia was log 6.38/ml and log 8.7/ml on first and 5th d.p.i. (died). Possible antigenic variation of strain on the third challenge or overdose of inocolum are postuled. Serum sample were collected during experiment for after evaluation. We verificated protection conferred against homologous T. evansi strain, as demonstred by others authors with T. congolense, T. brucei, T. cruzi and T. evansi. More studies are necessary to determinate which mechanisms are involved in the partial protection.
650 $aimmunization
650 $aPantanal
650 $aTrypanosoma evansi
650 $aImunização
653 $aInfeccao letal
653 $aLethal infection
700 1 $aSILVA, R. A. M. S.
700 1 $aRAMIREZ, L.
773 $tIn: CONGRESSO DA SOCIEDADE BRASILEIRA DE PARASITOLOGIA, 14., 1995, Goiania. Resumos... Goiania: [s.n.] 1995. p.325.
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