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Registro Completo |
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Biblioteca(s): |
Embrapa Clima Temperado. |
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Data corrente: |
16/10/2023 |
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Data da última atualização: |
16/10/2023 |
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Tipo da produção científica: |
Artigo em Periódico Indexado |
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Autoria: |
SOUZA, R. S. de; MARTINS, C. R.; CASTRO, C. M.; PINHEIRO, N. L.; MELLO-FARIAS, P. C. de. |
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Afiliação: |
RAFAELA SCHMIDT DE SOUZA; CARLOS ROBERTO MARTINS, CPACT; CAROLINE MARQUES CASTRO, CPACT; NATERCIA LOBATO PINHEIRO LIMA, CPACT; PAULO CELSO DE MELLO-FARIAS, UNIVERSIDADE FEDERAL DE PELOTAS. |
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Título: |
Genetic variability in pecan genotypes in Brazil. |
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Ano de publicação: |
2023 |
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Fonte/Imprenta: |
Revista Ceres, Viçosa, v. 70, n. 5, e70504, Aug./Sep 2023. |
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DOI: |
10.1590/0034-737X202370050004 |
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Idioma: |
Inglês |
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Conteúdo: |
Pecan crops has been expanding in recent years, mainly in southern Brazil. Genotypes that compose Brazilian orchards come from the USA and from selections of plants made by Brazilian producers. This study aimed to characterize with microsatellite markers pecan cultivars registered for cultivation in Brazil, including some selections made in the country. It is important to know the genetic variability of the pecan tree, as it facilitates the identification of possible phytotechnical deficiencies due to the genetic similarity between the plants, in addition to helping in the conservation of the species, among other. Thirty-four, out of forty collected accessions, were genotyped with 11 selected SSR (Simple Sequence Repeats) loci. Twenty-four polymorphic alleles were identified. The genetic similarity matrix, based on the Jaccard coefficient, ranged from 0.125 to 1.0; general mean of similarity was 0.46. The cluster analysis, which was carried out by the Unweighted Pair Group Method with Arithmetic Mean (UPGMA), classified pecan accessions into four main groups. Results showed that there is high genetic variability in germplasm evaluated, although some accessions may be duplicates. |
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Thesagro: |
Noz; Noz Peca. |
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Categoria do assunto: |
-- |
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URL: |
https://ainfo.cnptia.embrapa.br/digital/bitstream/doc/1157269/1/Artigo-2023-genetic-pecan.pdf
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Marc: |
LEADER 01782naa a2200205 a 4500
001 2157269
005 2023-10-16
008 2023 bl uuuu u00u1 u #d
024 7 $a10.1590/0034-737X202370050004$2DOI
100 1 $aSOUZA, R. S. de
245 $aGenetic variability in pecan genotypes in Brazil.$h[electronic resource]
260 $c2023
520 $aPecan crops has been expanding in recent years, mainly in southern Brazil. Genotypes that compose Brazilian orchards come from the USA and from selections of plants made by Brazilian producers. This study aimed to characterize with microsatellite markers pecan cultivars registered for cultivation in Brazil, including some selections made in the country. It is important to know the genetic variability of the pecan tree, as it facilitates the identification of possible phytotechnical deficiencies due to the genetic similarity between the plants, in addition to helping in the conservation of the species, among other. Thirty-four, out of forty collected accessions, were genotyped with 11 selected SSR (Simple Sequence Repeats) loci. Twenty-four polymorphic alleles were identified. The genetic similarity matrix, based on the Jaccard coefficient, ranged from 0.125 to 1.0; general mean of similarity was 0.46. The cluster analysis, which was carried out by the Unweighted Pair Group Method with Arithmetic Mean (UPGMA), classified pecan accessions into four main groups. Results showed that there is high genetic variability in germplasm evaluated, although some accessions may be duplicates.
650 $aNoz
650 $aNoz Peca
700 1 $aMARTINS, C. R.
700 1 $aCASTRO, C. M.
700 1 $aPINHEIRO, N. L.
700 1 $aMELLO-FARIAS, P. C. de
773 $tRevista Ceres, Viçosa$gv. 70, n. 5, e70504, Aug./Sep 2023.
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Registro original: |
Embrapa Clima Temperado (CPACT) |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Agricultura Digital. Para informações adicionais entre em contato com cnptia.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Agricultura Digital. |
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Data corrente: |
22/11/2012 |
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Data da última atualização: |
14/04/2020 |
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Tipo da produção científica: |
Artigo em Periódico Indexado |
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Circulação/Nível: |
A - 1 |
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Autoria: |
YOUNG, N. D.; JEX, A. R.; LI, B.; LIU, S.; YANG, L.; XIONG, Z.; LI, Y.; CANTACESSI, C.; HALL, R. S.; XU, X.; CHEN, F.; WU, X.; ZERLOTINI, A.; OLIVEIRA, G.; HOFMANN, A.; ZHANG, G.; FANG, X.; KANG, Y.; CAMPBELL, B. E.; LOUKAS, A.; RANGANATHAN, S.; ROLLINSON, D.; RINALDI, G.; BRINDLEY, P. J.; YANG, H.; WANG, J.; WANG, J.; GASSER, R. B. |
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Afiliação: |
NEIL D. YOUNG, The University of Melbourn; AARON R. JEX, The University of Melbourn; BO LI, BGI-Shenzhen; SHIPING LIU, BGI-Shenzhen; LINFENG YANG, BGI-Shenzhen; ZIJUN XIONG, BGI-Shenzhen; YIUNGRUI LI, BGI-Shenzhen; CINZIA CANTACESSI, The University of Melbourn; ROSS S. HALL, The University of Melbourn; XUN XU, BGI-Shenzhen; FANGYUAN CHEN, BGI-Shenzhen; XUAN WU, BGI-Shenzhen; ADHEMAR ZERLOTINI NETO, CNPTIA; GUILHERME OLIVEIRA, Instituto de Pesquisa René Rachou-Fiocruz; ANDREAS HOFMANN, The University of Melbourn, Griffith University; GUOJIE ZHANG, BGI-Shenzhen; XIAODONG FANG, BGI-Shenzhen; YI FANG, BGI-Shenzhen; BRONWYN E. CAMPBELL, The University of Melbourne; ALEX LOUKAS, James Cook University; SHOBA RANGANATHAN, Macquarie University, National University of Singapore; DAVID ROLLINSON, Natural History Museum, London; GABRIEL RINALDI, Universidad de la República, Montevideo, George Washington University; PAUL BRINDLEY, George Washington University; HUANMING YANG, BGI-Shenzhen; JUN WANG, BGI-Shenzhen; JIAN WANG, BGI-Shenzhen; ROBIN B. GASSER, The University of Melbourne. |
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Título: |
Whole-genome sequence of Schistosoma haematobium. |
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Ano de publicação: |
2012 |
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Fonte/Imprenta: |
Nature Genetics, v. 44, n. 2, p. 221-225, Feb. 2012. |
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DOI: |
10.1038/ng.1065 |
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Idioma: |
Inglês |
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Conteúdo: |
Schistosomiasis is a neglected tropical disease caused by blood flukes (genus Schistosoma; schistosomes) and affecting 200 million people worldwide1. No vaccines are available, and treatment relies on one drug, praziquantel2. Schistosoma haematobium has come into the spotlight as a major cause of urogenital disease, as an agent linked to bladder cancer1,3 and as a predisposing factor for HIV/AIDS4,5. The parasite is transmitted to humans from freshwater snails1. Worms dwell in blood vessels and release eggs that become embedded in the bladder wall to elicit chronic immune-mediated disease6 and induce squamous cell carcinoma7. Here we sequenced the 385-Mb genome of S. haematobium using Illumina-based technology at 74-fold coverage and compared it to sequences from related parasites8,9. We included genome annotation based on function, gene ontology, networking and pathway mapping. This genome now provides an unprecedented resource for many fundamental research areas and shows great promise for the design of new disease interventions. |
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Palavras-Chave: |
Sequência genômica. |
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Thesaurus NAL: |
Genome; Schistosoma haematobium. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 02258naa a2200493 a 4500
001 1940324
005 2020-04-14
008 2012 bl uuuu u00u1 u #d
024 7 $a10.1038/ng.1065$2DOI
100 1 $aYOUNG, N. D.
245 $aWhole-genome sequence of Schistosoma haematobium.$h[electronic resource]
260 $c2012
520 $aSchistosomiasis is a neglected tropical disease caused by blood flukes (genus Schistosoma; schistosomes) and affecting 200 million people worldwide1. No vaccines are available, and treatment relies on one drug, praziquantel2. Schistosoma haematobium has come into the spotlight as a major cause of urogenital disease, as an agent linked to bladder cancer1,3 and as a predisposing factor for HIV/AIDS4,5. The parasite is transmitted to humans from freshwater snails1. Worms dwell in blood vessels and release eggs that become embedded in the bladder wall to elicit chronic immune-mediated disease6 and induce squamous cell carcinoma7. Here we sequenced the 385-Mb genome of S. haematobium using Illumina-based technology at 74-fold coverage and compared it to sequences from related parasites8,9. We included genome annotation based on function, gene ontology, networking and pathway mapping. This genome now provides an unprecedented resource for many fundamental research areas and shows great promise for the design of new disease interventions.
650 $aGenome
650 $aSchistosoma haematobium
653 $aSequência genômica
700 1 $aJEX, A. R.
700 1 $aLI, B.
700 1 $aLIU, S.
700 1 $aYANG, L.
700 1 $aXIONG, Z.
700 1 $aLI, Y.
700 1 $aCANTACESSI, C.
700 1 $aHALL, R. S.
700 1 $aXU, X.
700 1 $aCHEN, F.
700 1 $aWU, X.
700 1 $aZERLOTINI, A.
700 1 $aOLIVEIRA, G.
700 1 $aHOFMANN, A.
700 1 $aZHANG, G.
700 1 $aFANG, X.
700 1 $aKANG, Y.
700 1 $aCAMPBELL, B. E.
700 1 $aLOUKAS, A.
700 1 $aRANGANATHAN, S.
700 1 $aROLLINSON, D.
700 1 $aRINALDI, G.
700 1 $aBRINDLEY, P. J.
700 1 $aYANG, H.
700 1 $aWANG, J.
700 1 $aWANG, J.
700 1 $aGASSER, R. B.
773 $tNature Genetics$gv. 44, n. 2, p. 221-225, Feb. 2012.
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