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Registro Completo |
Biblioteca(s): |
Embrapa Pantanal. |
Data corrente: |
11/01/2005 |
Data da última atualização: |
11/01/2005 |
Autoria: |
ABREU, U. G. P. de; SILVA, R. A. M. S.; BARROS, A. T. |
Afiliação: |
Embrapa Pantanal (Corumbá, MS). |
Título: |
Avaliação do controle da anemia infecciosa eqüina em fazendas na Sub-região da Nhecolândia, Pantanal Sul-Mato-Grossense. |
Ano de publicação: |
2004 |
Fonte/Imprenta: |
In: SIMPÓSIO SOBRE RECURSOS NATURAIS E SÓCIO-ECONÔMICOS DO PANTANAL, 4., 2004, Corumbá, MS. Sustentabilidade regional: anais. Corumbá: Embrapa Pantanal: UCDB: UFMS: SEBRAE-MS, 2004. (CD-ROM). |
Idioma: |
Português |
Conteúdo: |
A Anemia Infecciosa Eqüina (AIE) é causada por um retrovírus pertencente à subfamília dos lentivírus, o qual infectam membros da família Equidae. A AIE compromete irreversivelmente o desempenho dos eqüídeos, afetando indiretamente a pecuária extensiva e, sendo até o momento, uma doença incurável. Considera-se que, no Pantanal, a AIE se encontre disseminada, tendo causado grande mortalidade quando da
sua entrada na região em meados da década de 70. Durante seis anos, entre 1990 e 1995, a Embrapa Pantanal monitorou rebanhos de eqüídeos em 28 fazendas na subregião da Nhecolândia, com a realização de exames periódicos visando a identificação e separação dos animais positivos. Utilizando análise de fatores e de tipologia, o presente trabalho teve o objetivo de analisar o controle alcançado pelo manejo dos eqüídeos nas fazendas nas quais foi implantado o Programa de Prevenção e Controle de AIE, preconizado pela Embrapa Pantanal. Através da estimativa dos escores fatoriais, as fazendas foram distribuídas em cinco grupos. Foi observado que, em todas as fazendas onde foi efetivamente adotado, o Programa proporcionou o controle da AIE. |
Palavras-Chave: |
Anemia infecciosa equina; Controle; Monitoramento; Nhecolândia. |
Thesagro: |
Cavalo; Doença. |
Thesaurus Nal: |
Pantanal. |
Categoria do assunto: |
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Marc: |
LEADER 01989naa a2200229 a 4500 001 1811486 005 2005-01-11 008 2004 bl uuuu u00u1 u #d 100 1 $aABREU, U. G. P. de 245 $aAvaliação do controle da anemia infecciosa eqüina em fazendas na Sub-região da Nhecolândia, Pantanal Sul-Mato-Grossense. 260 $c2004 520 $aA Anemia Infecciosa Eqüina (AIE) é causada por um retrovírus pertencente à subfamília dos lentivírus, o qual infectam membros da família Equidae. A AIE compromete irreversivelmente o desempenho dos eqüídeos, afetando indiretamente a pecuária extensiva e, sendo até o momento, uma doença incurável. Considera-se que, no Pantanal, a AIE se encontre disseminada, tendo causado grande mortalidade quando da sua entrada na região em meados da década de 70. Durante seis anos, entre 1990 e 1995, a Embrapa Pantanal monitorou rebanhos de eqüídeos em 28 fazendas na subregião da Nhecolândia, com a realização de exames periódicos visando a identificação e separação dos animais positivos. Utilizando análise de fatores e de tipologia, o presente trabalho teve o objetivo de analisar o controle alcançado pelo manejo dos eqüídeos nas fazendas nas quais foi implantado o Programa de Prevenção e Controle de AIE, preconizado pela Embrapa Pantanal. Através da estimativa dos escores fatoriais, as fazendas foram distribuídas em cinco grupos. Foi observado que, em todas as fazendas onde foi efetivamente adotado, o Programa proporcionou o controle da AIE. 650 $aPantanal 650 $aCavalo 650 $aDoença 653 $aAnemia infecciosa equina 653 $aControle 653 $aMonitoramento 653 $aNhecolândia 700 1 $aSILVA, R. A. M. S. 700 1 $aBARROS, A. T. 773 $tIn: SIMPÓSIO SOBRE RECURSOS NATURAIS E SÓCIO-ECONÔMICOS DO PANTANAL, 4., 2004, Corumbá, MS. Sustentabilidade regional: anais. Corumbá: Embrapa Pantanal: UCDB: UFMS: SEBRAE-MS, 2004. (CD-ROM).
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Registro Completo
Biblioteca(s): |
Embrapa Soja. |
Data corrente: |
20/12/2005 |
Data da última atualização: |
13/01/2006 |
Autoria: |
PAGLIARINI, M. S.; BIONE, N. C. P.; BOLDRINI, K. R.; ALMEIDA, L. A. |
Título: |
A male-sterile mutation in soybean (Glycine max ) affecting chromosome arrangement in metaphase plate and cytokinesis. |
Ano de publicação: |
2005 |
Fonte/Imprenta: |
In: CONGRESSO BRASILEIRO DE GENÉTICA, 51., 2005, Águas de Lindóia. Resumos... Águas de Lindóia: SBG, 2005. |
Descrição Física: |
1 CD-ROM. |
Idioma: |
Inglês |
Notas: |
Seção: Genética e Melhoramento - Resumos: Pdf.579. |
Conteúdo: |
In autogamous crop species with small flowers, such as soybean, manual cross-pollination to produce large quantities of hybrid seed is difficult and tedious. A number of molecular approaches have been used to study and regulate male sterility on various agricultural crops for F1 hybrid production. On the other hand, there are many plant species that display natural mutants that cause male sterility. Mutant nuclear genes (Mendelian inheritance) affecting male cell and organ development are designated ms genes. They
usually are recessive and typically expressed in specific sporophytic tissues at different stages. Several soybean male-sterile, female-sterile mutant lines have been described genetically and cytologically. Spontaneous male-sterile,female-fertile mutations have been identified in Brazilian lines of soybean breeding program developed by Embrapa Soybean. Sterile plants were identified, flower buds were for meiotic analysis and fixed in FAA (3 ethanol 95%:1 acetic acid:1 formaldehyde)for 24 h; transferred to 70%alcohol and stored at 4 0 C until use.Microsporocytes were prepared by squashing and stained with 0.5% acetic carmine.Nine male-sterile plants were cytologically analyzed.Pollen grains were stained with 0.5%acetic carmine and starch content was evaluated with lugol.In these male-sterile,female-fertile mutant bivalent arrangement at the metaphase plate and cytokinesis were affected.Until diakinesis, meiosis was perfectly normal with chromosome pairing as bivalents. From this phase, in several meiocytes, bivalents were not able to organize a single metaphase plate and remained scattered in the cytoplasm in a few or several groups. In these meiocytes, chromosomes segregated in both divisions giving rise to several micronuclei. However, the main cause of male sterility was the absence of cytokinesis after telophase II. Instead of the typical tetrads of microspores, four nucleate coenocytic microspores were formed. Typical tetrads of microspores were not detected in the present mutant.Instead of four haploid microspores, the meiotic products were replaced by four-nucleate coenocytic microspores.After pollen wall formation,and without suffering the pollen mitoses, the coenocytic microspores underwent gradual engorgement by starch,and entered into a progressive process of degeneration. Pollen sterility was complete. A ratio of a 3 male-fertile:1 male-sterile plant was found among self-pollinated progeny in line BR98-197H,where the mutation originally appeared and within segregating families. At least three male-sterile mutant lines identified in the Brazilian breeding
program and critically analyzed in the cytological context presented differential characteristics from those reported for a specific ms mutation. All of them were related to abnormal cytokinesis after telophase II, but always with some additional characteristics. Data suggest that some loci controlling male fertility in soybean tend more towards spontaneous mutations than others. MenosIn autogamous crop species with small flowers, such as soybean, manual cross-pollination to produce large quantities of hybrid seed is difficult and tedious. A number of molecular approaches have been used to study and regulate male sterility on various agricultural crops for F1 hybrid production. On the other hand, there are many plant species that display natural mutants that cause male sterility. Mutant nuclear genes (Mendelian inheritance) affecting male cell and organ development are designated ms genes. They
usually are recessive and typically expressed in specific sporophytic tissues at different stages. Several soybean male-sterile, female-sterile mutant lines have been described genetically and cytologically. Spontaneous male-sterile,female-fertile mutations have been identified in Brazilian lines of soybean breeding program developed by Embrapa Soybean. Sterile plants were identified, flower buds were for meiotic analysis and fixed in FAA (3 ethanol 95%:1 acetic acid:1 formaldehyde)for 24 h; transferred to 70%alcohol and stored at 4 0 C until use.Microsporocytes were prepared by squashing and stained with 0.5% acetic carmine.Nine male-sterile plants were cytologically analyzed.Pollen grains were stained with 0.5%acetic carmine and starch content was evaluated with lugol.In these male-sterile,female-fertile mutant bivalent arrangement at the metaphase plate and cytokinesis were affected.Until diakinesis, meiosis was perfectly normal with chromosome pairing as bivale... Mostrar Tudo |
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LEADER 03653naa a2200181 a 4500 001 1468657 005 2006-01-13 008 2005 bl uuuu u00u1 u #d 100 1 $aPAGLIARINI, M. S. 245 $aA male-sterile mutation in soybean (Glycine max ) affecting chromosome arrangement in metaphase plate and cytokinesis. 260 $c2005 300 $c1 CD-ROM. 500 $aSeção: Genética e Melhoramento - Resumos: Pdf.579. 520 $aIn autogamous crop species with small flowers, such as soybean, manual cross-pollination to produce large quantities of hybrid seed is difficult and tedious. A number of molecular approaches have been used to study and regulate male sterility on various agricultural crops for F1 hybrid production. On the other hand, there are many plant species that display natural mutants that cause male sterility. Mutant nuclear genes (Mendelian inheritance) affecting male cell and organ development are designated ms genes. They usually are recessive and typically expressed in specific sporophytic tissues at different stages. Several soybean male-sterile, female-sterile mutant lines have been described genetically and cytologically. Spontaneous male-sterile,female-fertile mutations have been identified in Brazilian lines of soybean breeding program developed by Embrapa Soybean. Sterile plants were identified, flower buds were for meiotic analysis and fixed in FAA (3 ethanol 95%:1 acetic acid:1 formaldehyde)for 24 h; transferred to 70%alcohol and stored at 4 0 C until use.Microsporocytes were prepared by squashing and stained with 0.5% acetic carmine.Nine male-sterile plants were cytologically analyzed.Pollen grains were stained with 0.5%acetic carmine and starch content was evaluated with lugol.In these male-sterile,female-fertile mutant bivalent arrangement at the metaphase plate and cytokinesis were affected.Until diakinesis, meiosis was perfectly normal with chromosome pairing as bivalents. From this phase, in several meiocytes, bivalents were not able to organize a single metaphase plate and remained scattered in the cytoplasm in a few or several groups. In these meiocytes, chromosomes segregated in both divisions giving rise to several micronuclei. However, the main cause of male sterility was the absence of cytokinesis after telophase II. Instead of the typical tetrads of microspores, four nucleate coenocytic microspores were formed. Typical tetrads of microspores were not detected in the present mutant.Instead of four haploid microspores, the meiotic products were replaced by four-nucleate coenocytic microspores.After pollen wall formation,and without suffering the pollen mitoses, the coenocytic microspores underwent gradual engorgement by starch,and entered into a progressive process of degeneration. Pollen sterility was complete. A ratio of a 3 male-fertile:1 male-sterile plant was found among self-pollinated progeny in line BR98-197H,where the mutation originally appeared and within segregating families. At least three male-sterile mutant lines identified in the Brazilian breeding program and critically analyzed in the cytological context presented differential characteristics from those reported for a specific ms mutation. All of them were related to abnormal cytokinesis after telophase II, but always with some additional characteristics. Data suggest that some loci controlling male fertility in soybean tend more towards spontaneous mutations than others. 700 1 $aBIONE, N. C. P. 700 1 $aBOLDRINI, K. R. 700 1 $aALMEIDA, L. A. 773 $tIn: CONGRESSO BRASILEIRO DE GENÉTICA, 51., 2005, Águas de Lindóia. Resumos... Águas de Lindóia: SBG, 2005.
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